M
Michael O. Hengartner
Researcher at University of Zurich
Publications - 61
Citations - 6619
Michael O. Hengartner is an academic researcher from University of Zurich. The author has contributed to research in topics: Caenorhabditis elegans & Programmed cell death. The author has an hindex of 29, co-authored 49 publications receiving 6053 citations. Previous affiliations of Michael O. Hengartner include University of Virginia & Stony Brook University.
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Classification of cell death: recommendations of the Nomenclature Committee on Cell Death
Guido Kroemer,Guido Kroemer,Guido Kroemer,Lorenzo Galluzzi,Lorenzo Galluzzi,Lorenzo Galluzzi,Peter Vandenabeele,John M. Abrams,Emad S. Alnemri,Eric H. Baehrecke,Mikhail V. Blagosklonny,Wafik S. El-Deiry,Pierre Golstein,Pierre Golstein,Douglas R. Green,Michael O. Hengartner,Richard A. Knight,Sharad Kumar,Stuart A. Lipton,Stuart A. Lipton,Stuart A. Lipton,Walter Malorni,Gabriel Núñez,Marcus E. Peter,Juerg Tschopp,Junying Yuan,Mauro Piacentini,Boris Zhivotovsky,Gerry Melino,Gerry Melino +29 more
TL;DR: This study details the 2009 recommendations of the NCCD on the use of cell death-related terminology including ‘entosis’, ‘mitotic catastrophe”,’ ‘necrosis‚ ‘necroptosis‚’ and ‘pyroptotic’.
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Engulfment genes cooperate with ced-3 to promote cell death in Caenorhabditis elegans
TL;DR: It is shown that blocking engulfment enhances cell survival when cells are subjected to weak pro-apoptotic signals, indicating that genes that mediate corpse removal can also function to actively kill cells.
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Developmental apoptosis in C. elegans: a complex CEDnario.
TL;DR: New insights into the regulation and execution of apoptosis in C. elegans have been made and will undoubtedly influence the perception of developmental apoptotic in more complex species, including humans.
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Caenorhabditis elegans inhibitor of apoptosis protein (iap) homologue bir-1 plays a conserved role in cytokinesis
TL;DR: It is suggested that BIRPs may regulate cytoskeletal changes in diverse biological processes including cytokinesis and apoptosis and may play roles in other, as yet unidentified, cellular processes besides apoptosis.
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Candidate Adaptor Protein CED-6 Promotes the Engulfment of Apoptotic Cells in C. elegans
TL;DR: The cloning and functional characterization of ced-6, a gene specifically required for the engulfment of apoptotic cells in the nematode C. elegans, are reported and suggest that CED-6 is an adaptor molecule acting in a signal transduction pathway that specifically mediates the recognition and engulfing of apoptosis cells.