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Motoaki Sano

Researcher at Keio University

Publications -  250
Citations -  14298

Motoaki Sano is an academic researcher from Keio University. The author has contributed to research in topics: Heart failure & Medicine. The author has an hindex of 55, co-authored 230 publications receiving 12192 citations. Previous affiliations of Motoaki Sano include Tokai University & Chugai Pharmaceutical Co..

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Cardiomyocytes can be generated from marrow stromal cells in vitro

TL;DR: Analysis of the isoform of contractile protein genes, such as myosin heavy chain, myos in light chain, and alpha-actin, indicated that their muscle phenotype was similar to that of fetal ventricular cardiomyocytes.
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Distinct metabolic flow enables large-scale purification of mouse and human pluripotent stem cell-derived cardiomyocytes.

TL;DR: A nongenetic method for mass-producing cardiomyocytes from mouse and human PSC derivatives that is based on the marked biochemical differences in glucose and lactate metabolism between cardiomers and noncardiomyocyte and could facilitate progress toward PSC-based cardiac regenerative therapy.
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Temporal dynamics of cardiac immune cell accumulation following acute myocardial infarction.

TL;DR: Timely reperfusion reduced the total number of leukocytes accumulated in the post-MI period, shifting the peak of innate immune response towards earlier and blunting the wave of adaptive immune response.
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Inhalation of hydrogen gas reduces infarct size in the rat model of myocardial ischemia-reperfusion injury.

TL;DR: Inhalation of H(2) gas is promising strategy to alleviate ischemia-reperfusion injury coincident with recanalization of coronary artery, thereby preventing deleterious left ventricular remodeling.
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Crosstalk between Glucocorticoid Receptor and Nutritional Sensor mTOR in Skeletal Muscle

TL;DR: It is shown that direct target genes of the glucocorticoid receptor (GR) in skeletal muscle, i.e., REDD1 and KLF15 inhibits mTOR activity, but via a distinct mechanism involving BCAT2 gene activation, and GR is a liaison involving a variety of downstream molecular cascades toward muscle atrophy.