M
Myriam Foglietta
Researcher at University of Turin
Publications - 33
Citations - 1151
Myriam Foglietta is an academic researcher from University of Turin. The author has contributed to research in topics: Immune system & Interleukin 21. The author has an hindex of 16, co-authored 30 publications receiving 890 citations. Previous affiliations of Myriam Foglietta include University of Texas MD Anderson Cancer Center.
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Journal ArticleDOI
COVID-19 severity and mortality in patients with chronic lymphocytic leukemia: a joint study by ERIC, the European Research Initiative on CLL, and CLL Campus.
Lydia Scarfò,Thomas Chatzikonstantinou,Gian Matteo Rigolin,Giulia Quaresmini,Marina Motta,Candida Vitale,José A. García-Marco,Jose Angel Hernandez-Rivas,Fatima Miras,Mónica Baile,Juan Marquet,Carsten Utoft Niemann,Gianluigi Reda,Talha Munir,Eva Gimeno,Monia Marchetti,Francesca Maria Quaglia,Marzia Varettoni,Julio Delgado,Sunil Iyengar,Ann Janssens,Roberto Marasca,Angela Ferrari,Carolina Cuéllar-García,Gilad Itchaki,Martin Spacek,Lorenzo De Paoli,Luca Laurenti,Mark-David Levin,Enrico Lista,Francesca Romana Mauro,Martin Simkovic,Ellen van der Spek,Elisabeth Vandenberghe,Livio Trentin,Ewa Wasik-Szczepanek,Rosa Ruchlemer,Dominique Bron,Maria Rosaria De Paolis,Giovanni Del Poeta,Lucia Farina,Myriam Foglietta,Massimo Gentile,Yair Herishanu,Tobias Herold,Ozren Jakšić,Arnon P. Kater,Sabina Kersting,Lara Malerba,Lorella Orsucci,Viola Maria Popov,Paolo Sportoletti,Mohamed A. Yassin,Barbara Pocali,Gábor Barna,Annalisa Chiarenza,Gimena Dos Santos,Eugene Nikitin,Martin Andres,Maria Dimou,Michael Doubek,Alicia Enrico,Yervand K Hakobyan,Olga Kalashnikova,Macarena Ortiz Pareja,Maria Papaioannou,Davide Rossi,Nimish Shah,Amit Shrestha,Oana Stanca,Niki Stavroyianni,Vladimir Strugov,Constantine S. Tam,Mihnea Zdrenghea,Marta Coscia,Kostas Stamatopoulos,Giuseppe Rossi,Alessandro Rambaldi,Emili Montserrat,Robin Foà,Antonio Cuneo,Paolo Ghia +81 more
TL;DR: In CLL, COVID-19 severity increases with age; antileukemic treatment (particularly BTK inhibitors) appears to exert a protective effect; age and comorbidities did not impact on mortality, alluding to a relevant role of CLL and immunodeficiency.
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Effector gammadelta T cells and tumor cells as immune targets of zoledronic acid in multiple myeloma.
Sara Mariani,M Muraro,Francesca Pantaleoni,F. Fiore,Barbara Nuschak,Silvia Peola,Myriam Foglietta,Antonio Palumbo,Marta Coscia,Barbara Castella,Benedetto Bruno,Raffaello Bertieri,L Boano,Mario Boccadoro,Massimo Massaia +14 more
TL;DR: Zol induced antimyeloma activity through the monocyte-dependent activation of γδ T cells and by enhancing the immunosensitivity of myeloma cells to γ Δ T cells, and Mevastatin, a specific inhibitor of hydroxy-methylglutaryl-CoA reductase, completely abrogated this antimyelin activity.
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Enhanced ability of dendritic cells to stimulate innate and adaptive immunity on short-term incubation with zoledronic acid.
F. Fiore,Barbara Castella,Barbara Nuschak,Raffaello Bertieri,Sara Mariani,Benedetto Bruno,Francesca Pantaleoni,Myriam Foglietta,Mario Boccadoro,Massimo Massaia +9 more
TL;DR: Large numbers of gammadelta T cells with effector and costimulatory activities are rapidly generated by Zol-treated iDC/mDC, and this strategy is worthy of further investigation to improve adoptive cell therapy and vaccine interventions against tumors and infections.
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Immune Modulation by Zoledronic Acid in Human Myeloma: An Advantageous Cross-Talk between Vγ9Vδ2 T Cells, αβ CD8+ T Cells, Regulatory T Cells, and Dendritic Cells
Barbara Castella,Chiara Riganti,F. Fiore,Francesca Pantaleoni,Maria Elisa Canepari,Silvia Peola,Myriam Foglietta,Antonio Palumbo,Amalia Bosia,Marta Coscia,Mario Boccadoro,Massimo Massaia +11 more
TL;DR: ZA has multiple immune modulatory activities that allow MM DC to effectively handle the concurrent activation of γδ T cells and MHC-restricted CD8+ αβ antitumor effector T cells.
Journal ArticleDOI
IGHV unmutated CLL B cells are more prone to spontaneous apoptosis and subject to environmental prosurvival signals than mutated CLL B cells.
Marta Coscia,Francesca Pantaleoni,Chiara Riganti,Candida Vitale,Micol Maria Rigoni,Silvia Peola,Barbara Castella,Myriam Foglietta,Valentina Griggio,Daniela Drandi,Marco Ladetto,Amalia Bosia,Mario Boccadoro,Massimo Massaia +13 more
TL;DR: Despite their more aggressive features, UM CLL B cells are more susceptible to spontaneous apoptosis and depend from environmental prosurvival signals, which can be exploited as a selective target of therapeutic interventions.