M
Myung-Sook Choi
Researcher at Kyungpook National University
Publications - 324
Citations - 15714
Myung-Sook Choi is an academic researcher from Kyungpook National University. The author has contributed to research in topics: Insulin resistance & Cholesterol. The author has an hindex of 60, co-authored 324 publications receiving 13590 citations. Previous affiliations of Myung-Sook Choi include Korea Institute of Science and Technology & Chungnam National University.
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Obesity and Its Metabolic Complications: The Role of Adipokines and the Relationship between Obesity, Inflammation, Insulin Resistance, Dyslipidemia and Nonalcoholic Fatty Liver Disease
Un Ju Jung,Myung-Sook Choi +1 more
TL;DR: This review focuses on the role of several adipokines associated with obesity and the potential impact on obesity-related metabolic diseases.
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Chlorogenic acid exhibits anti-obesity property and improves lipid metabolism in high-fat diet-induced-obese mice
TL;DR: It is suggested that caffeic acid and chlorogenic acid improve body weight, lipid metabolism and obesity-related hormones levels in high-fat fed mice.
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Effect of citrus flavonoids on lipid metabolism and glucose-regulating enzyme mRNA levels in type-2 diabetic mice.
TL;DR: The current results suggest that hesperidin and naringin are beneficial for improving hyperlipidemia and hyperglycemia in type-2 diabetic animals by partly regulating the fatty acid and cholesterol metabolism and affecting the gene expression of glucose-regulating enzymes.
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Plasma and Hepatic Cholesterol and Hepatic Activities of 3-Hydroxy-3-methyl-glutaryl-CoA Reductase and Acyl CoA: Cholesterol Transferase Are Lower in Rats Fed Citrus Peel Extract or a Mixture of Citrus Bioflavonoids
Song Hae Bok,Sung Heui Lee,Yong Bok Park,KiHwan Bae,Kwang Hee Son,Tae Sook Jeong,Myung-Sook Choi +6 more
TL;DR: The inhibition of HMG-CoA reductase and ACAT activities resulting from the supplementation of either tangerine-peel extract or a combination of its bioflavonoids could account for the decrease in fecal neutral sterol that appears to compensate for the decreased cholesterol biosynthesis in the liver.
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Mitochondrial ROS govern the LPS-induced pro-inflammatory response in microglia cells by regulating MAPK and NF-κB pathways
Junghyung Park,Ju-Sik Min,Bokyung Kim,Unbin Chae,Jong Won Yun,Myung-Sook Choi,Il-Keun Kong,Kyu-Tae Chang,Dong-Seok Lee +8 more
TL;DR: Results indicate that mitochondria constitute a major source of ROS generation in LPS-mediated activated microglia cells and suggest that a potential strategy in the development of therapy for inflammation-associated degenerative neurological diseases involves targeting the regulation of mitochondrial ROS in microglial cells.