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Patrick May

Researcher at University of Luxembourg

Publications -  177
Citations -  6861

Patrick May is an academic researcher from University of Luxembourg. The author has contributed to research in topics: Gene & Epilepsy. The author has an hindex of 37, co-authored 149 publications receiving 5188 citations. Previous affiliations of Patrick May include Max Planck Society & Zuse Institute Berlin.

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Journal ArticleDOI

Mercator: a fast and simple web server for genome scale functional annotation of plant sequence data

TL;DR: The Mercator pipeline automatically assigns functional terms to protein or nucleotide sequences using the MapMan 'BIN' ontology, which is tailored for functional annotation of plant 'omics' data.
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Identification of Nutrient-Responsive Arabidopsis and Rapeseed MicroRNAs by Comprehensive Real-Time Polymerase Chain Reaction Profiling and Small RNA Sequencing

TL;DR: In this paper, a quantitative real-time polymerase chain reaction platform was used to detect phosphorus (P) or nitrogen (N) status-responsive pri-miR species.
Journal ArticleDOI

Integrated multi-omics of the human gut microbiome in a case study of familial type 1 diabetes

TL;DR: This work presents an integrative approach to resolve the taxonomic and functional attributes of gastrointestinal microbiota at the metagenomic, metatranscriptomic and metaproteomic levels and applies it to samples from four families with multiple cases of type 1 diabetes mellitus.
Book ChapterDOI

ZIB structure prediction pipeline: composing a complex biological workflow through web services

TL;DR: This case study demonstrates the ability of an easy orchestration of complex biological workflows based on Web services as building blocks and Triana as workflow engine.
Journal ArticleDOI

De novo loss-or gain-of-function mutations in KCNA2 cause epileptic encephalopathy

TL;DR: Next-generation sequencing results establish KCNA2 as a new gene involved in human neurodevelopmental disorders through two different mechanisms, predicting either hyperexcitability or electrical silencing of KV1.2-expressing neurons.