P
Payal Kapur
Researcher at University of Texas Southwestern Medical Center
Publications - 244
Citations - 11224
Payal Kapur is an academic researcher from University of Texas Southwestern Medical Center. The author has contributed to research in topics: Clear cell renal cell carcinoma & Cancer. The author has an hindex of 42, co-authored 223 publications receiving 8743 citations. Previous affiliations of Payal Kapur include University of Texas at Dallas & University of California, Berkeley.
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Journal ArticleDOI
Characterization of Inflammation in Syphilitic Villitis and in Villitis of Unknown Etiology
Payal Kapur,Dinesh Rakheja,Ana M. Gomez,Jeanne S. Sheffield,Pablo J. Sánchez,Beverly Barton Rogers +5 more
TL;DR: Comparing the maternal vs. fetal origin of the infiltrating lymphocytes in VUE and syphilitic villitis and the immunophenotypic data provided by previous studies indicate that the immune response in both syphiliaria and VUE is similar, raising the possibility of a similar immunopathogenetic pathway.
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Fatty acid synthase expression in cutaneous melanocytic neoplasms.
TL;DR: Of interest, congenital melanocytic nevi showed strong fatty acid synthase expression, similar to that seen in metastatic melanoma, which may represent persistence of or regression to a fetal phenotype since normal fetal tissues are known to express high levels of fatty Acid synthase.
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IFNγ-Induced IFIT5 Promotes Epithelial-to-Mesenchymal Transition in Prostate Cancer via miRNA Processing.
U-Ging Lo,Rey-Chen Pong,Diane Yang,Leah Gandee,Elizabeth Hernandez,Andrew Dang,Chung Jung Lin,John Santoyo,Shihong Ma,Rajni Sonavane,Jun Huang,Shu-Fen Tseng,Loredana Moro,Arnaldo A. Arbini,Payal Kapur,Ganesh V. Raj,Dalin He,Chih Ho Lai,Ho Lin,Jer Tsong Hsieh,Jer Tsong Hsieh +20 more
TL;DR: A unique IFIT5-XRN1 complex involved in the turnover of specific tumor suppressive microRNAs is the underlying mechanism of IFNγ-induced epithelial-to-mesenchymal transition in prostate cancer.
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Slug, a unique androgen-regulated transcription factor, coordinates androgen receptor to facilitate castration resistance in prostate cancer.
Kaijie Wu,Crystal Gore,Lin Yang,Ladan Fazli,Martin E. Gleave,Rey Chen Pong,Guanghua Xiao,Linlin Zhang,Eun-Jin Yun,Shu Fen Tseng,Payal Kapur,Dalin He,Jer Tsong Hsieh +12 more
TL;DR: There is a reciprocal regulation between Slug and AR not only in transcriptional regulation but also in protein bioactivity, and Slug-AR complex plays an important role in accelerating the androgen-independent outgrowth of CRPC.
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Loss of histone H3 lysine 36 trimethylation is associated with an increased risk of renal cell carcinoma-specific death.
Thai H. Ho,Payal Kapur,Richard W. Joseph,Daniel J. Serie,Jeanette E. Eckel-Passow,Pan Tong,Jing Wang,Erik P. Castle,Melissa L. Stanton,John C. Cheville,Eric Jonasch,James Brugarolas,Alexander S. Parker +12 more
TL;DR: Although SETD2 DNA and mRNA alterations are not associated with overall survival, evidence is provided that deregulation of the H3K36me3 axis is associated with a higher risk of renal cell carcinoma-specific death.