R
R Selley
Publications - 2
Citations - 685
R Selley is an academic researcher. The author has contributed to research in topics: Genome & Mutagenesis (molecular biology technique). The author has an hindex of 2, co-authored 2 publications receiving 679 citations.
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A systematic, genome-wide, phenotype-driven mutagenesis programme for gene function studies in the mouse.
Patrick M. Nolan,Josephine Peters,Mark A. Strivens,Derek C. Rogers,Jim J. Hagan,Nigel K. Spurr,Ian C. Gray,Lucie Vizor,D Brooker,E Whitehill,R Washbourne,Tertius Hough,Simon Greenaway,M Hewitt,Xue Zhong Liu,Stefan L. McCormack,K Pickford,R Selley,Christine A. Wells,Zuzanna Tymowska-Lalanne,P. Roby,Peter H. Glenister,C Thornton,Caroline Thaung,J A Stevenson,Ruth M. Arkell,Philomena Mburu,Rachel E. Hardisty,Amy E. Kiernan,Alexandra Erven,Karen P. Steel,Stéphanie Voegeling,Jean-Louis Guénet,Carole D. Nickols,R Sadri,M Nasse,Adrian M. Isaacs,Kay E. Davies,M Browne,Elizabeth M. C. Fisher,Joanne E. Martin,Sohaila Rastan,Steve D.M. Brown,Jackie Hunter +43 more
TL;DR: A genome-wide, phenotype-driven screen for dominant mutations in the mouse is undertaken, which has led to a substantial increase in themouse mutant resource and represents a first step towards systematic studies of gene function in mammalian genetics.
Journal ArticleDOI
Towards a mutant map of the mouse – new models of neurological, behavioural, deafness, bone, renal and blood disorders
Sohaila Rastan,Tertius Hough,A. Kierman,Rachel E. Hardisty,Alexandra Erven,I. C. Gray,S. Voeling,Adrian M. Isaacs,H. Tsai,Mark A. Strivens,R Washbourne,C Thornton,Simon Greenaway,M Hewitt,S. McCormick,R Selley,Christine A. Wells,Zuzanna Tymowska-Lalanne,P. Roby,Philomena Mburu,Derek C. Rogers,Jim J. Hagan,Charlie Reavill,Kay E. Davies,Peter H. Glenister,Elizabeth M. C. Fisher,Joanne E. Martin,Lucie Vizor,M. Bouzyk,David P. Kelsell,Jean-Louis Guénet,Karen P. Steel,Steven A. Sheardown,Nigel K. Spurr,Ian C. Gray,Josephine Peters,P Nolan,A. J. Hunter,Steve D.M. Brown +38 more
TL;DR: This project has used ENU mutagenesis in the mouse for the rapid generation of novel mutant phenotypes for use as animal models of human disease and for gene function assignment, and can extrapolate that it has recovered over 700 mutants from the screening programme.