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Rizwan Masood

Researcher at University of Southern California

Publications -  75
Citations -  3708

Rizwan Masood is an academic researcher from University of Southern California. The author has contributed to research in topics: Angiogenesis & Vascular endothelial growth factor. The author has an hindex of 33, co-authored 75 publications receiving 3581 citations. Previous affiliations of Rizwan Masood include Charité & State University of New York System.

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Vascular endothelial growth factor (VEGF) is an autocrine growth factor for VEGF receptor–positive human tumors

TL;DR: VEGF is an autocrine growth factor for tumor cell lines that express VEGFRs and it is shown that inhibition of VEGF (VEGF antisense oligonucleotide AS-3) or V EGFRs (neutralizing antibodies) inhibited the proliferation of these cell lines in vitro.
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Vascular endothelial growth factor/vascular permeability factor is an autocrine growth factor for AIDS–Kaposi sarcoma

TL;DR: This study shows that AIDS-KS cell lines express higher levels of vascular endothelial growth factor/vascular permeability factor (VEGF/VGF) than either human umbilical vein endothelial cells or human aortic smooth muscle cells, and shows that VEGF is an autocrine growth factor for AIDS- KS cells.
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Seroprevalence of human herpesvirus-8 (HHV-8) in countries of Southeast Asia compared to the USA, the Caribbean and Africa

TL;DR: Nasopharyngeal and oral carcinoma patients from Malaysia, Hong Kong and Sri Lanka who have very high EBV titres show that there is little, if any, cross-reactivity between antibodies to these two gamma viruses, suggesting that human herpes virus-8 (HHV-8) may be either a recently introduced virus or one that has extremely low infectivity.
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Interleukin-10 is an autocrine growth factor for acquired immunodeficiency syndrome-related B-cell lymphoma.

TL;DR: Evaluation of expression of IL-10 in tumor-derived B-cell lines and primary tumor cells from patients with AIDS-lymphoma cells suggests an autocrine growth mechanism for IL-20 in AIDS-related lymphoma cells and that IL- 10 may be important in its pathogenesis.