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Serge Marty

Researcher at French Institute of Health and Medical Research

Publications -  34
Citations -  2984

Serge Marty is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Hippocampal formation & GABAergic. The author has an hindex of 23, co-authored 32 publications receiving 2857 citations. Previous affiliations of Serge Marty include Centre national de la recherche scientifique & Max Planck Society.

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Three-dimensional architecture of presynaptic terminal cytomatrix.

TL;DR: 3D analysis reveals the morphological constraints exerted by the presynaptic molecular scaffold and links SVs are tightly interconnected in the axonal bouton, and this network is preferentially connected to the AZ.
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GABAergic stimulation regulates the phenotype of hippocampal interneurons through the regulation of brain-derived neurotrophic factor.

TL;DR: Gamma-Aminobutyric acid switches from enhancing to repressing brain-derived neurotrophic factor mRNA synthesis during the maturation of hippocampal neurons in vitro, and treatment with muscimol and BDNF induced a decrease in cell size and NPY immunoreactivity of interneurons.
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Neuronal activity and brain-derived neurotrophic factor regulate the density of inhibitory synapses in organotypic slice cultures of postnatal hippocampus.

TL;DR: Investigation of organotypic slice cultures in postnatal day 7 rats found that neuronal activity regulates the density of inhibitory synapses made by postnatal hippocampal interneurons, and BDNF could mediate part of this regulation.
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GABAergic stimulation switches from enhancing to repressing BDNF expression in rat hippocampal neurons during maturation in vitro

TL;DR: Observations support the hypothesis that GABA might have neurotrophic effects on embryonic or perinatal hippocampal neurons, which are mediated by BDNF.
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Neurotrophins and activity-dependent plasticity of cortical interneurons

TL;DR: It is hypothesized that neurotrophins might mediate some of the actions of neuronal activity on GABA-containing neurons, because neuronal activity fails to upregulate the expression of neuropeptide Y in hippocampal cultures from BDNF-deficient mice.