S
Steven H. Strogatz
Researcher at Cornell University
Publications - 227
Citations - 92888
Steven H. Strogatz is an academic researcher from Cornell University. The author has contributed to research in topics: Josephson effect & Kuramoto model. The author has an hindex of 79, co-authored 219 publications receiving 85750 citations. Previous affiliations of Steven H. Strogatz include Boston College & Purdue University.
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Journal ArticleDOI
Quantifying the benefits of vehicle pooling with shareability networks
Paolo Santi,Giovanni Resta,Michael Szell,Stanislav Sobolevsky,Steven H. Strogatz,Carlo Ratti +5 more
TL;DR: The notion of shareability network is introduced, which allows to model the collective benefits of sharing as a function of passenger inconvenience, and to efficiently compute optimal sharing strategies on massive datasets, and demonstrates the feasibility of a shareable taxi service in New York City.
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Modeling a synthetic multicellular clock: Repressilators coupled by quorum sensing
TL;DR: The effect of coupling through intercell signaling in a population of Escherichia coli cells expressing a synthetic biological clock is studied to predict that a diverse and noisy community of such genetic oscillators interacting through a quorum-sensing mechanism should self-synchronize in a robust way, leading to a substantially improved global rhythmicity in the system.
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Solvable model for chimera states of coupled oscillators
TL;DR: The first exact results about the stability, dynamics, and bifurcations of chimera states are obtained by analyzing a minimal model consisting of two interacting populations of oscillators.
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Time Delay in the Kuramoto Model of Coupled Oscillators
TL;DR: In this paper, the authors generalize the Kuramoto model of coupled oscillators to allow time-delayed interactions and derive exact formulas for the stability boundaries of the incoherent and synchronized states, as a function of the delay.
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Cellular Construction of a Circadian Clock: Period Determination in the Suprachiasmatic Nuclei
TL;DR: The results show that circadian period in the whole animal is determined by averaging widely dispersed periods of individual clock cells, and demonstrate that the tau mutation affects circadian function in a cell-autonomous manner.