Susan J. Nixon

TL;DR: It is concluded that PTRF-Cavin is required for caveola formation and sequestration of mobile caveolin into immobile caveolae.

TL;DR: Using quantitative light microscopy and a modified immunoelectron microscopic technique, the entry pathway of the cholera toxin binding subunit (CTB) in primary embryonic fibroblasts is characterized and the major carriers involved in initial entry of CTB were identified as uncoated tubular or ring-shaped structures.

TL;DR: Results show that a Rab protein localizes directly to the monolayer surface of LDs, suggesting that recruitment of Rab18 is regulated by the metabolic state of individual LDs.

TL;DR: Cavin-4 is expressed predominantly in muscle, and its distribution is perturbed in human muscle disease associated with Caveolin-3 dysfunction, identifying Cavin-4 as a novel muscle disease candidate caveolar protein.

TL;DR: Cav1–/– mice exhibited impaired liver regeneration and low survival after a partial hepatectomy and treatment with glucose drastically increased survival and reestablished progression of the cell cycle.