Xinfen Yu

TL;DR: Observations suggest the province-specific H9N2 virus gene pools increase the genetic diversity of H7N9 via dynamic reassortments and also imply that G0 has not gained overwhelming fitness and the virus continues to undergo reassortment.

TL;DR: Examination of poultry and environmental specimens from local markets and farms in Hangzhou provides direct evidence that H9N2 strains coexisted with the novel human-pathogenic H7N9 influenza virus in epidemiologically linked live poultry markets.

TL;DR: The sequences of the novel avian-origin H7N9 influenza viruses from Hangzhou City contained important amino acid substitutions related to human adaptation, which might be under adaptation pressure that will help it “jump” from avian to human hosts.

TL;DR: The emergence of pMRV150-like or pIP1202-like plasmids in many bacterial pathogens and nonpathogens occupying diverse niches with global geographical distribution indicates an increasing risk to public health worldwide.

TL;DR: The acute respiratory infection and pharyngoconjunctival fever outbreak in Hangzhou in 2011 were caused by the completely homologous type 3 adenovirus in subgenus B.