Example of Open Biology format
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Example of Open Biology format Example of Open Biology format Example of Open Biology format Example of Open Biology format Example of Open Biology format Example of Open Biology format Example of Open Biology format
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Example of Open Biology format Example of Open Biology format Example of Open Biology format Example of Open Biology format Example of Open Biology format Example of Open Biology format Example of Open Biology format
Sample paper formatted on SciSpace - SciSpace
This content is only for preview purposes. The original open access content can be found here.
open access Open Access

Open Biology — Template for authors

Publisher: The Royal Society
Categories Rank Trend in last 3 yrs
Biochemistry, Genetics and Molecular Biology (all) #25 of 204 up up by 19 ranks
Neuroscience (all) #19 of 110 up up by 23 ranks
Immunology #43 of 202 up up by 51 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 438 Published Papers | 3695 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 03/07/2020
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Related Journals

open access Open Access
recommended Recommended

PLOS

Quality:  
High
CiteRatio: 11.0
SJR: 4.127
SNIP: 2.005
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 5.3
SJR: 1.515
SNIP: 0.684
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 10.1
SJR: 2.615
SNIP: 1.649
open access Open Access
recommended Recommended

BMJ Publishing Group

Quality:  
High
CiteRatio: 28.7
SJR: 6.333
SNIP: 4.294

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

4.931

27% from 2018

Impact factor for Open Biology from 2016 - 2019
Year Value
2019 4.931
2018 3.89
2017 3.286
2016 3.481
graph view Graph view
table view Table view

8.4

12% from 2019

CiteRatio for Open Biology from 2016 - 2020
Year Value
2020 8.4
2019 7.5
2018 6.0
2017 5.0
2016 7.1
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 27% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 12% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

3.078

30% from 2019

SJR for Open Biology from 2016 - 2020
Year Value
2020 3.078
2019 2.368
2018 2.549
2017 2.082
2016 2.929
graph view Graph view
table view Table view

1.477

25% from 2019

SNIP for Open Biology from 2016 - 2020
Year Value
2020 1.477
2019 1.177
2018 1.008
2017 0.875
2016 1.199
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 30% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 25% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Open Biology

Guideline source: View

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The Royal Society

Open Biology

Open Biology is a selective, online journal that welcomes original, high-quality research in cell and developmental biology, molecular and structural biology, biochemistry, neuroscience, immunology, microbiology and genetics. Articles submitted to Open Biology benefit from its...... Read More

Biochemistry, Genetics and Molecular Biology

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Last updated on
03 Jul 2020
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ISSN
2046-2441
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Impact Factor
High - 1.25
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Open Access
No
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Sherpa RoMEO Archiving Policy
Green faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
Vancouver
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent con-version. Phys Rev B. 1982;25(7):4515–4532. Available from: 10.1103/PhysRevB.25.4515.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1098/RSOB.120080
PINK1 is activated by mitochondrial membrane potential depolarization and stimulates Parkin E3 ligase activity by phosphorylating Serine 65
01 May 2012 - Open Biology

Abstract:

Missense mutations in PTEN-induced kinase 1 (PINK1) cause autosomal-recessive inherited Parkinson's disease (PD). We have exploited our recent discovery that recombinant insect PINK1 is catalytically active to test whether PINK1 directly phosphorylates 15 proteins encoded by PD-associated genes as well as proteins reported to... Missense mutations in PTEN-induced kinase 1 (PINK1) cause autosomal-recessive inherited Parkinson's disease (PD). We have exploited our recent discovery that recombinant insect PINK1 is catalytically active to test whether PINK1 directly phosphorylates 15 proteins encoded by PD-associated genes as well as proteins reported to bind PINK1. We have discovered that insect PINK1 efficiently phosphorylates only one of these proteins, namely the E3 ligase Parkin. We have mapped the phosphorylation site to a highly conserved residue within the Ubl domain of Parkin at Ser65. We show that human PINK1 is specifically activated by mitochondrial membrane potential (Δψm) depolarization, enabling it to phosphorylate Parkin at Ser65. We further show that phosphorylation of Parkin at Ser65 leads to marked activation of its E3 ligase activity that is prevented by mutation of Ser65 or inactivation of PINK1. We provide evidence that once activated, PINK1 autophosphorylates at several residues, including Thr257, which is accompanied by an electrophoretic mobility band-shift. These results provide the first evidence that PINK1 is activated following Δψm depolarization and suggest that PINK1 directly phosphorylates and activates Parkin. Our findings indicate that monitoring phosphorylation of Parkin at Ser65 and/or PINK1 at Thr257 represent the first biomarkers for examining activity of the PINK1-Parkin signalling pathway in vivo. Our findings also suggest that small molecule activators of Parkin that mimic the effect of PINK1 phosphorylation may confer therapeutic benefit for PD. read more read less

Topics:

Parkin (69%)69% related to the paper, PINK1 (58%)58% related to the paper, Phosphorylation (52%)52% related to the paper, Ubiquitin ligase (51%)51% related to the paper
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753 Citations
open accessOpen access Journal Article DOI: 10.1098/RSOB.170070
Progress and prospects of early detection in lung cancer
Sean Knight, Phil A. Crosbie1, Haval Balata, Jakub Chudziak2, Tracy Hussell2, Caroline Dive2, Caroline Dive1
01 Sep 2017 - Open Biology

Abstract:

Lung cancer is the leading cause of cancer-related death in the world. It is broadly divided into small cell (SCLC, approx. 15% cases) and non-small cell lung cancer (NSCLC, approx. 85% cases). The main histological subtypes of NSCLC are adenocarcinoma and squamous cell carcinoma, with the presence of specific DNA mutations a... Lung cancer is the leading cause of cancer-related death in the world. It is broadly divided into small cell (SCLC, approx. 15% cases) and non-small cell lung cancer (NSCLC, approx. 85% cases). The main histological subtypes of NSCLC are adenocarcinoma and squamous cell carcinoma, with the presence of specific DNA mutations allowing further molecular stratification. If identified at an early stage, surgical resection of NSCLC offers a favourable prognosis, with published case series reporting 5-year survival rates of up to 70% for small, localized tumours (stage I). However, most patients (approx. 75%) have advanced disease at the time of diagnosis (stage III/IV) and despite significant developments in the oncological management of late stage lung cancer over recent years, survival remains poor. In 2014, the UK Office for National Statistics reported that patients diagnosed with distant metastatic disease (stage IV) had a 1-year survival rate of just 15–19% compared with 81–85% for stage I. read more read less

Topics:

Survival rate (60%)60% related to the paper, Lung cancer (58%)58% related to the paper, Adenocarcinoma (53%)53% related to the paper
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544 Citations
open accessOpen access Journal Article DOI: 10.1098/RSOB.180002
Targeting BCL-2 regulated apoptosis in cancer
Kirsteen J. Campbell1, Stephen W.G. Tait1
01 May 2018 - Open Biology

Abstract:

The ability of a cell to undergo mitochondrial apoptosis is governed by pro- and anti-apoptotic members of the BCL-2 protein family. The equilibrium of pro- versus anti-apoptotic BCL-2 proteins ensures appropriate regulation of programmed cell death during development and maintains organismal health. When unbalanced, the BCL-... The ability of a cell to undergo mitochondrial apoptosis is governed by pro- and anti-apoptotic members of the BCL-2 protein family. The equilibrium of pro- versus anti-apoptotic BCL-2 proteins ensures appropriate regulation of programmed cell death during development and maintains organismal health. When unbalanced, the BCL-2 family can act as a barrier to apoptosis and facilitate tumour development and resistance to cancer therapy. Here we discuss the BCL-2 family, their deregulation in cancer and recent pharmaceutical developments to target specific members of this family as cancer therapy. read more read less

Topics:

Bcl-2 family (59%)59% related to the paper, Cancer (55%)55% related to the paper, Protein family (53%)53% related to the paper, Programmed cell death (53%)53% related to the paper
View PDF
358 Citations
open accessOpen access Journal Article DOI: 10.1098/RSOB.200223
Wound healing: cellular mechanisms and pathological outcomes.
Holly N. Wilkinson1, Matthew J. Hardman1
30 Sep 2020 - Open Biology

Abstract:

Wound healing is a complex, dynamic process supported by a myriad of cellular events that must be tightly coordinated to efficiently repair damaged tissue. Derangement in wound-linked cellular behaviours, as occurs with diabetes and ageing, can lead to healing impairment and the formation of chronic, non-healing wounds. These... Wound healing is a complex, dynamic process supported by a myriad of cellular events that must be tightly coordinated to efficiently repair damaged tissue. Derangement in wound-linked cellular behaviours, as occurs with diabetes and ageing, can lead to healing impairment and the formation of chronic, non-healing wounds. These wounds are a significant socioeconomic burden due to their high prevalence and recurrence. Thus, there is an urgent requirement for the improved biological and clinical understanding of the mechanisms that underpin wound repair. Here, we review the cellular basis of tissue repair and discuss how current and emerging understanding of wound pathology could inform future development of efficacious wound therapies. read more read less

Topics:

Wound healing (50%)50% related to the paper
View PDF
351 Citations
open accessOpen access Journal Article DOI: 10.1098/RSOB.120139
T-cell epitope vaccine design by immunoinformatics
Atanas Patronov1, Irini Doytchinova1
01 Jan 2013 - Open Biology

Abstract:

Vaccination is generally considered to be the most effective method of preventing infectious diseases. All vaccinations work by presenting a foreign antigen to the immune system in order to evoke an immune response. The active agent of a vaccine may be intact but inactivated (‘attenuated’) forms of the causative pathogens (ba... Vaccination is generally considered to be the most effective method of preventing infectious diseases. All vaccinations work by presenting a foreign antigen to the immune system in order to evoke an immune response. The active agent of a vaccine may be intact but inactivated (‘attenuated’) forms of the causative pathogens (bacteria or viruses), or purified components of the pathogen that have been found to be highly immunogenic. The increased understanding of antigen recognition at molecular level has resulted in the development of rationally designed peptide vaccines. The concept of peptide vaccines is based on identification and chemical synthesis of B-cell and T-cell epitopes which are immunodominant and can induce specific immune responses. The accelerating growth of bioinformatics techniques and applications along with the substantial amount of experimental data has given rise to a new field, called immunoinformatics. Immunoinformatics is a branch of bioinformatics dealing with in silico analysis and modelling of immunological data and problems. Different sequence- and structure-based immunoinformatics methods are reviewed in the paper. read more read less

Topics:

Epitope (53%)53% related to the paper, Antigen (52%)52% related to the paper
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328 Citations
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SciSpace is a very innovative solution to the formatting problem and existing providers, such as Mendeley or Word did not really evolve in recent years.

- Andreas Frutiger, Researcher, ETH Zurich, Institute for Biomedical Engineering

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What to expect from SciSpace?

Speed and accuracy over MS Word

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With SciSpace, you do not need a word template for Open Biology.

It automatically formats your research paper to The Royal Society formatting guidelines and citation style.

You can download a submission ready research paper in pdf, LaTeX and docx formats.

Time comparison

Time taken to format a paper and Compliance with guidelines

Plagiarism Reports via Turnitin

SciSpace has partnered with Turnitin, the leading provider of Plagiarism Check software.

Using this service, researchers can compare submissions against more than 170 million scholarly articles, a database of 70+ billion current and archived web pages. How Turnitin Integration works?

Turnitin Stats
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Freedom from formatting guidelines

One editor, 100K journal formats – world's largest collection of journal templates

With such a huge verified library, what you need is already there.

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Easy support from all your favorite tools

Open Biology format uses Vancouver citation style.

Automatically format and order your citations and bibliography in a click.

SciSpace allows imports from all reference managers like Mendeley, Zotero, Endnote, Google Scholar etc.

Frequently asked questions

1. Can I write Open Biology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Open Biology guidelines and auto format it.

2. Do you follow the Open Biology guidelines?

Yes, the template is compliant with the Open Biology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Open Biology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Open Biology citation style.

4. Can I use the Open Biology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Open Biology.

5. Can I use a manuscript in Open Biology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Open Biology that you can download at the end.

6. How long does it usually take you to format my papers in Open Biology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Open Biology.

7. Where can I find the template for the Open Biology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Open Biology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Open Biology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Open Biology an online tool or is there a desktop version?

SciSpace's Open Biology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Open Biology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Open Biology?”

11. What is the output that I would get after using Open Biology?

After writing your paper autoformatting in Open Biology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Open Biology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Open Biology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Open Biology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Open Biology?

The 5 most common citation types in order of usage for Open Biology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Open Biology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Open Biology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Open Biology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Open Biology Endnote style according to Elsevier guidelines.

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Typset automatically formats your research paper to Open Biology formatting guidelines and citation style.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

Andreas Frutiger
Researcher & Ex MS Word user
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