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Journal ArticleDOI

Cyclic estradiol treatment phasically potentiates endogenous cholecystokinin's satiating action in ovariectomized rats.

Lori Asarian, +1 more
- 01 May 1999 - 
- Vol. 20, Iss: 4, pp 445-450
TLDR
Results suggest that a phasic potentiation of the endogenous CCK satiety-signalling system is part of the mechanism for the decrease in meal size in female rats during estrus.
About
This article is published in Peptides.The article was published on 1999-05-01. It has received 77 citations till now. The article focuses on the topics: Estradiol benzoate & Devazepide.

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Citations
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Journal ArticleDOI

The role of estrogens in control of energy balance and glucose homeostasis.

TL;DR: The literature in both rodents and humans on the role of estrogens and their receptors in the control of energy homeostasis and glucose metabolism in health and metabolic diseases is reviewed.
Journal ArticleDOI

Modulation of appetite by gonadal steroid hormones

TL;DR: A variety of peripheral feedback controls of eating, including ghrelin, cholecystokinin (CCK), glucagon, hepatic fatty acid oxidation, insulin and leptin, has been shown to be estradiol-sensitive under at least some conditions and may mediate the estrogenic inhibition of eating.
Journal ArticleDOI

Sex differences in the physiology of eating

TL;DR: The variety and physiological importance of what has been learned so far warrant intensifying basic, translational, and clinical research on sex differences in eating.
Journal ArticleDOI

Cyclic Estradiol Treatment Normalizes Body Weight and Restores Physiological Patterns of Spontaneous Feeding and Sexual Receptivity in Ovariectomized Rats

TL;DR: These results provide the first demonstration that the induction of a cyclic, near-physiological pattern of plasma estradiol is sufficient to maintain normal levels of body weight, spontaneous feeding patterns, total food intake, and (together with progesterone) sexual receptivity in ovariectomized rats.
Journal ArticleDOI

Central effects of estradiol in the regulation of food intake, body weight, and adiposity

TL;DR: Men and post-menopausal women accumulate more fat in their intra-abdominal depots than pre- menopausal women, resulting in a greater risk of developing complications associated with obesity.
References
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Book

Manual of histologic staining methods of the Armed forces institute of pathology

Lee G Luna
TL;DR: This work states that stem cells: mechanisms of inflammation,” Annual Review of Pathology, vol.
Journal ArticleDOI

Gonadal hormones and behavioral regulation of body weight

TL;DR: Gonadal hormones have important effects on the behaviors that determine body weight in laboratory rats, and may act directly on separate neural loci to inhibit food intake and stimulate locomotor activity, possibly by lowering the set-point of a hypothalamic lipostat.
Journal ArticleDOI

Some effects of ovarian hormones on food intake and body weight in female rats.

TL;DR: Estradiol-treated ovariectomized rats are far more responsive to the weight- and appetite-promoting actions of progesterone than are ovariECTomized-adrenalectomization rats, suggesting that the principal action of progestersone on energy balance may be to interfere with the effects of estradiol.
Journal ArticleDOI

Ovarian influences on the meal patterns of female rats.

TL;DR: The results suggest that the transient changes in food intake caused by estradiol withdrawal and replacement are accomplished by permanent changes in meal size followed by compensatory changes in the number of meals consumed per day.
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