Docosahexaenoic acid dietary supplementation enhances the effects of exercise on synaptic plasticity and cognition.
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TLDR
The results indicate that the DHA diet enhanced the effects of exercise on cognition and BDNF-related synaptic plasticity, a capacity that may be used to promote mental health and reduce risk of neurological disorders.About:
This article is published in Neuroscience.The article was published on 2008-08-26 and is currently open access. It has received 276 citations till now. The article focuses on the topics: Physical exercise & Brain-derived neurotrophic factor.read more
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Journal ArticleDOI
Brain foods: the effects of nutrients on brain function
TL;DR: Understanding the molecular basis of the effects of food on cognition will help to determine how best to manipulate diet in order to increase the resistance of neurons to insults and promote mental fitness.
Journal ArticleDOI
Exercise and the brain: something to chew on.
TL;DR: Optimal maintenance of brain health might depend on exercise and intake of natural products, and the potential synergy between diet and exercise could involve common cellular pathways important for neurogenesis, cell survival, synaptic plasticity and vascular function.
OtherDOI
The Influence of Exercise on Cognitive Abilities
TL;DR: Data are suggestive that aerobic fitness enhances cognitive strategies enabling to respond effectively to an imposed challenge with a better yield in task performance.
Journal ArticleDOI
Long-chain polyunsaturated fatty acids (LCPUFA) from genesis to senescence: The influence of LCPUFA on neural development, aging, and neurodegeneration
TL;DR: This review shows that LCPUFA are essential throughout life, especially for neuronal membrane integrity and function, and also contribute in prevention of brain hypoperfusion.
Journal ArticleDOI
Towards Establishing Dietary Reference Intakes for Eicosapentaenoic and Docosahexaenoic Acids
William S. Harris,Dariush Mozaffarian,Michael Lefevre,Cheryl D. Toner,John Colombo,Stephen C. Cunnane,Joanne M. Holden,David M. Klurfeld,Martha Clare Morris,Jay Whelan +9 more
TL;DR: The Technical Committee on Dietary Lipids of the International Life Sciences Institute North America sponsored a workshop to consider whether the body of evidence specific to the major chronic diseases in the United States--coronary heart disease, cancer, and cognitive decline--had evolved sufficiently to justify reconsideration of DRI for EPA+.
References
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Neurotrophins and neuronal plasticity.
TL;DR: A role for NTs as selective retrograde messengers that regulate synaptic efficacy is suggested, based on evidence that NT synthesis is rapidly regulated by neuronal activity and that NTs are released in an activity-dependent manner from neuronal dendrites.
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Physical activity and risk of cognitive impairment and dementia in elderly persons.
TL;DR: Regular physical activity could represent an important and potent protective factor for cognitive decline and dementia in elderly persons.
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Long-Lasting Neurotrophin-Induced Enhancement of Synaptic Transmission in the Adult Hippocampus
Hyejin Kang,Erin M. Schuman +1 more
TL;DR: Long-term potentiation could still be elicited in slices previously potentiated by exposure to the neurotrophic factors, which implies that these two forms of plasticity may use at least partially independent cellular mechanisms.
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Ageing, fitness and neurocognitive function.
Arthur F. Kramer,Sowon Hahn,Neal J. Cohen,Marie T. Banich,Edward McAuley,Catherine R. Harrison,Julie Chason,Eli Vakil,Lynn Bardell,Richard A. Boileau,Angela Colcombe +10 more
TL;DR: It is found that those who received aerobic training showed substantial improvements in performance on tasks requiring executive control compared with anaerobically trained subjects.
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Hippocampal BDNF mediates the efficacy of exercise on synaptic plasticity and cognition
TL;DR: An association between CREB and BDNF expression and cognitive function, such that animals who were the fastest learners and had the best recall showed the highest expression of BDNF and associated CREB mRNA levels, suggest a functional role for CREB under the control ofBDNF in mediating the exercise‐induced enhancement in learning and memory.