Fine particulate matter (PM2.5): The culprit for chronic lung diseases in China.
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TLDR
This review will discuss the pathophysiology of PM2.5 in respiratory diseases, which are helpful for the prevention of air pollution and treatment of respiratory tract inflammatory diseases.About:
This article is published in Chronic Diseases and Translational Medicine.The article was published on 2018-08-28 and is currently open access. It has received 128 citations till now.read more
Citations
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Impact of COVID-19 lockdown on NO2, O3, PM2.5 and PM10 concentrations and assessing air quality changes in Baghdad, Iraq
TL;DR: The results of NO2 tropospheric column extracted from the Sentinel-5P satellite shown the NO2 emissions reduced up to 35 to 40% across Iraq, due to lockdown measures, between January and July 2020, especially across the major cities such as Baghdad, Basra and Erbil.
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Air pollution by NO2 and PM2.5 explains COVID-19 infection severity by overexpression of angiotensin-converting enzyme 2 in respiratory cells: a review
TL;DR: Overall, it is demonstrated that there is a link between NO 2 emissions, PM 2.5 levels, ACE-2 expression and COVID-19 infection severity, and air pollution should be reduced in places where confirmed cases of CO VID-19 are unexpectedly high.
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Air pollution aggravating COVID-19 lethality? Exploration in Asian cities using statistical models.
Ankit Gupta,Ankit Gupta,Hemant Bherwani,Hemant Bherwani,Sneha Gautam,Saima Anjum,Kavya Musugu,Narendra Kumar,Avneesh Anshul,Rakesh Kumar,Rakesh Kumar +10 more
TL;DR: It is suggested that there exists a positive correlation between the level of air pollution of a region and the lethality related to COVID-19, indicating air pollution to be an elemental and concealed factor in aggravating the global burden of deaths related to the disease.
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Environmental Exposures and Asthma Development: Autophagy, Mitophagy, and Cellular Senescence.
Karan Sachdeva,Danh C. Do,Yan Zhang,Yan Zhang,Xinyue Hu,Xinyue Hu,Jingsi Chen,Jingsi Chen,Peisong Gao +8 more
TL;DR: Environmental pollutants and allergens induce oxidative stress and mitochondrial dysfunction, leading to key features of allergic asthma, and understanding the regulatory mechanisms of autophagy, mitophagy and cellular senescence may allow for the development of new therapeutic targets for asthma.
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Short-term exposure to ambient air quality of the most polluted Indian cities due to lockdown amid SARS-CoV-2.
TL;DR: Reporting of the possible recovery extent in air quality may help to guide alternative policy intervention in form of short term lockdown so as to testify whether this type of unconventional policy decisions may be put forward to attain a green environment.
References
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Effect of ambient PM(2.5) on lung mitochondrial damage and fusion/fission gene expression in rats.
Ruijin Li,Xiaojing Kou,Hong Geng,Jingfang Xie,Zhenhua Yang,Yuexia Zhang,Zongwei Cai,Chuan Dong +7 more
TL;DR: It is suggested that the pathological injury observed in rat lung exposed to PM2.5 is associated with mitochondrial fusion-fission dysfunction, ROS generation, mitochondrial lipid peroxidation, and cellular homeostasis imbalance.
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Air pollution and multiple acute respiratory outcomes
Annunziata Faustini,Massimo Stafoggia,Paola Colais,Giovanna Berti,Luigi Bisanti,Ennio Cadum,Achille Cernigliaro,Sandra Mallone,Corrado Scarnato,Francesco Forastiere +9 more
TL;DR: Age, sex and previous ischaemic heart disease acted as effect modifiers for different outcomes and the temporal relationship between the pollutant increases and hospitalisations or mortality for respiratory diseases differs.
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Air pollution affects lung cancer survival
Sandrah P. Eckel,Myles Cockburn,Yu-Hsiang Shu,Huiyu Deng,Frederick Lurmann,Lihua Liu,Frank D. Gilliland +6 more
TL;DR: The hypothesis that air pollution exposures after lung cancer diagnosis shorten survival is supported, and the largest associations were for patients with early-stage non-small cell cancers, particularly adenocarcinomas.
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Instillation of particulate matter 2.5 induced acute lung injury and attenuated the injury recovery in ACE2 knockout mice.
Chung I. Lin,Chin Hung Tsai,Yu Ling Sun,Wen Yeh Hsieh,Yi Chang Lin,Cheng Yi Chen,Cheng Yi Chen,Chih-Sheng Lin +7 more
TL;DR: The results hint that PM2.5 could induce severe ALI through pulmonary inflammation, and the repair after acute PM2,5 postinstillation could be attenuated in the absence of ACE2.
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Air particulate matter induced oxidative stress and inflammation in cardiovascular disease and atherosclerosis: The role of Nrf2 and AhR-mediated pathways
TL;DR: Data supporting the role of oxidative stress and inflammation in PM-induced cardiovascular diseases and atherosclerosis and the importance of Nrf2-and AhR- dependent regulatory pathways in the PM- induced cardiovascular events and Atherosclerosis are focused on.
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