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Open AccessJournal ArticleDOI

Fine particulate matter (PM2.5): The culprit for chronic lung diseases in China.

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TLDR
This review will discuss the pathophysiology of PM2.5 in respiratory diseases, which are helpful for the prevention of air pollution and treatment of respiratory tract inflammatory diseases.
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This article is published in Chronic Diseases and Translational Medicine.The article was published on 2018-08-28 and is currently open access. It has received 128 citations till now.

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Journal ArticleDOI

Impact of COVID-19 lockdown on NO2, O3, PM2.5 and PM10 concentrations and assessing air quality changes in Baghdad, Iraq

TL;DR: The results of NO2 tropospheric column extracted from the Sentinel-5P satellite shown the NO2 emissions reduced up to 35 to 40% across Iraq, due to lockdown measures, between January and July 2020, especially across the major cities such as Baghdad, Basra and Erbil.
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Air pollution by NO2 and PM2.5 explains COVID-19 infection severity by overexpression of angiotensin-converting enzyme 2 in respiratory cells: a review

TL;DR: Overall, it is demonstrated that there is a link between NO 2 emissions, PM 2.5 levels, ACE-2 expression and COVID-19 infection severity, and air pollution should be reduced in places where confirmed cases of CO VID-19 are unexpectedly high.
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Air pollution aggravating COVID-19 lethality? Exploration in Asian cities using statistical models.

TL;DR: It is suggested that there exists a positive correlation between the level of air pollution of a region and the lethality related to COVID-19, indicating air pollution to be an elemental and concealed factor in aggravating the global burden of deaths related to the disease.
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Environmental Exposures and Asthma Development: Autophagy, Mitophagy, and Cellular Senescence.

TL;DR: Environmental pollutants and allergens induce oxidative stress and mitochondrial dysfunction, leading to key features of allergic asthma, and understanding the regulatory mechanisms of autophagy, mitophagy and cellular senescence may allow for the development of new therapeutic targets for asthma.
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Short-term exposure to ambient air quality of the most polluted Indian cities due to lockdown amid SARS-CoV-2.

TL;DR: Reporting of the possible recovery extent in air quality may help to guide alternative policy intervention in form of short term lockdown so as to testify whether this type of unconventional policy decisions may be put forward to attain a green environment.
References
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Journal ArticleDOI

Long-term ambient fine particulate matter air pollution and lung cancer in a large cohort of never-smokers.

TL;DR: The present findings strengthen the evidence that ambient concentrations of PM(2.5) measured in recent decades are associated with small but measurable increases in lung cancer mortality.
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Spatial and temporal variations of six criteria air pollutants in 31 provincial capital cities in China during 2013-2014.

TL;DR: Great impacts of coal combustion and biomass burning in the winter, long range transport of windblown dust in the spring, and secondary aerosol formation throughout the year are suggested by this study.
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Health burden attributable to ambient PM2.5 in China.

TL;DR: Air quality modeling and cost-benefits analysis of emission reduction scenarios and corresponding health benefits in meeting the site-specific annual PM2.5 concentrations in China are expected to be 24.0%, 44.8%, 70.8, and 85.2% of the total current mortalities when the PWA PM3.5 meets the WHO IT-1,IT-2, IT-3, and AQG, respectively.
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Systematic review of Chinese studies of short-term exposure to air pollution and daily mortality.

TL;DR: A meta-analysis on 33 time-series and case-crossover studies conducted in China to assess mortality effects of short-term exposure to particulate matter with aerodynamic diameters less than 10 and 2.5 μm found significant associations between air pollution exposure and increased mortality risks.
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PM2.5-induced oxidative stress triggers autophagy in human lung epithelial A549 cells

TL;DR: The results suggest the possibility that PM2.5-induced oxidative stress probably plays a key role in autophagy in A549 cells, which may contribute to PM3.0-induced impairment of pulmonary function.
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