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Frank D. Gilliland

Researcher at University of Southern California

Publications -  435
Citations -  38398

Frank D. Gilliland is an academic researcher from University of Southern California. The author has contributed to research in topics: Population & Asthma. The author has an hindex of 90, co-authored 395 publications receiving 34549 citations. Previous affiliations of Frank D. Gilliland include University of Washington & National Institutes of Health.

Papers
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The effect of air pollution on lung development from 10 to 18 years of age.

TL;DR: Current levels of air pollution have chronic, adverse effects on lung development in children from the age of 10 to 18 years, leading to clinically significant deficits in attained FEV as children reach adulthood.
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Urinary and sexual function after radical prostatectomy for clinically localized prostate cancer : The Prostate Cancer Outcomes Study

TL;DR: This study suggests that radical prostatectomy is associated with significant erectile dysfunction and some decline in urinary function, and these results may be particularly helpful to community-based physicians and their patients with prostate cancer who face difficult treatment decisions.
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Asthma in exercising children exposed to ozone: a cohort study.

TL;DR: Investigation of the relation between newly-diagnosed asthma and team sports in a cohort of children exposed to different concentrations and mixtures of air pollutants found air pollution and outdoor exercise could contribute to the development of asthma in children.
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Meta-analysis of genome-wide association studies of asthma in ethnically diverse North American populations

Dara G. Torgerson, +79 more
- 01 Sep 2011 - 
TL;DR: The results suggest that some asthma susceptibility loci are robust to differences in ancestry when sufficiently large samples sizes are investigated, and that ancestry-specific associations also contribute to the complex genetic architecture of asthma.
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Ambient air pollution and atherosclerosis in Los Angeles.

TL;DR: These results represent the first epidemiologic evidence of an association between atherosclerosis and ambient air pollution and given the leading role of cardiovascular disease as a cause of death and the large populations exposed to ambient PM2.5, these findings may be important and need further confirmation.