A
Amy S. Shah
Researcher at Cincinnati Children's Hospital Medical Center
Publications - 145
Citations - 3055
Amy S. Shah is an academic researcher from Cincinnati Children's Hospital Medical Center. The author has contributed to research in topics: Type 2 diabetes & Diabetes mellitus. The author has an hindex of 24, co-authored 108 publications receiving 2116 citations. Previous affiliations of Amy S. Shah include University of Cincinnati Academic Health Center & Veterans Health Administration.
Papers
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Journal ArticleDOI
Proteomic diversity of high density lipoproteins: our emerging understanding of its importance in lipid transport and beyond
TL;DR: Key challenges facing the field are highlighted, particularly the need to identify and define the function of HDL subspecies to better inform attempts to pharmacologically manipulate HDL for the benefit of cardiovascular disease and possibly other maladies.
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Cardiovascular Disease Risk Factors in Youth With Diabetes Mellitus: A Scientific Statement From the American Heart Association
David M. Maahs,Stephen R. Daniels,Sarah D. de Ferranti,Helén L. Dichek,Joseph T. Flynn,Benjamin I. Goldstein,Aaron S. Kelly,Kristen J. Nadeau,Pamela Martyn-Nemeth,Stavroula K. Osganian,Laurie Quinn,Amy S. Shah,Elaine M. Urbina +12 more
TL;DR: This scientific statement summarizes and interprets guidelines and new developments in the field in the past decade and outlines future research and clinical needs to improve cardiovascular health and risk factor management in youth with diabetes mellitus.
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Androgens inhibit adipogenesis during human adipose stem cell commitment to preadipocyte formation.
Gregorio D. Chazenbalk,Prapti Singh,Dana Irge,Dana Irge,Amy S. Shah,David H. Abbott,David H. Abbott,Daniel A. Dumesic +7 more
TL;DR: The findings indicate that androgens, in part through androgen receptor action, impair BMP4-induced commitment of SC hASCs to preadipocytes and also reduce early-stage adipocyte differentiation, perhaps limiting adipocyte numbers and fat storage in SC abdominal adipose.
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Obesity is a significant risk factor for prostate cancer at the time of biopsy.
Stephen J. Freedland,Stephen J. Freedland,Joanne Wen,Joanne Wen,Melanie Wuerstle,Melanie Wuerstle,Amy S. Shah,Amy S. Shah,Dominic W Lai,Bita Moalej,Bita Moalej,Christina Atala,Christina Atala,William J. Aronson,William J. Aronson +14 more
TL;DR: The fact that current prostate cancer screening practices may be biased against obese men is supported, as obesity was not significantly associated with prostate cancer risk in this equal-access, clinic-based population.
Journal ArticleDOI
Fine-mapping, trans-ancestral and genomic analyses identify causal variants, cells, genes and drug targets for type 1 diabetes.
Catherine C. Robertson,Inshaw Jrj.,Suna Onengut-Gumuscu,Chen W-M.,D F Santa Cruz,Huan Yang,Antony J. Cutler,Crouch Djm.,Emily Farber,S L Bridges,Jeffrey C. Edberg,Robert P. Kimberly,Jane H. Buckner,Panagiotis Deloukas,Panagiotis Deloukas,Jasmin Divers,Dana Dabelea,Jean M. Lawrence,Santica M. Marcovina,Amy S. Shah,Carla J. Greenbaum,Mark A. Atkinson,Peter K. Gregersen,Jorge R. Oksenberg,Flemming Pociot,Flemming Pociot,Marian Rewers,Andrea K. Steck,David B. Dunger,Linda S. Wicker,Patrick Concannon,John A. Todd,Stephen S. Rich +32 more
TL;DR: The largest and most diverse genetic study of type 1 diabetes (T1D) to date (61,427 participants) yielded 78 genome-wide-significant (P < 5'5'×'10'8) regions, including 36 new regions as discussed by the authors.