A
Anita Sil
Researcher at University of California, San Francisco
Publications - 62
Citations - 3401
Anita Sil is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Gene & Histoplasma. The author has an hindex of 23, co-authored 45 publications receiving 2593 citations. Previous affiliations of Anita Sil include University of California, Berkeley & Howard Hughes Medical Institute.
Papers
More filters
Journal ArticleDOI
Amplification-free detection of SARS-CoV-2 with CRISPR-Cas13a and mobile phone microscopy.
Parinaz Fozouni,Sungmin Son,María Díaz de León Derby,Gavin J. Knott,Gavin J. Knott,Carley N. Gray,Carley N. Gray,Michael V. D’Ambrosio,Chunyu Zhao,Neil A. Switz,G. Renuka Kumar,G. Renuka Kumar,Stephanie I. Stephens,Stephanie I. Stephens,Daniela Boehm,Daniela Boehm,Chia-Lin Tsou,Chia-Lin Tsou,Jeffrey Shu,Jeffrey Shu,Abdul Bhuiya,Maxim Armstrong,Andrew R. Harris,Pei Yi Chen,Pei Yi Chen,Jeannette M. Osterloh,Anke Meyer-Franke,Bastian Joehnk,Keith Walcott,Anita Sil,Charles Langelier,Katherine S. Pollard,Emily D. Crawford,Andreas S. Puschnik,Maira Phelps,Amy Kistler,Joseph L. DeRisi,Jennifer A. Doudna,Daniel A. Fletcher,Melanie Ott +39 more
TL;DR: This work reports the development of an amplification-free CRISPR-Cas13a assay for direct detection of SARS-CoV-2 from nasal swab RNA that can be read with a mobile phone microscope and has the potential to enable rapid, low-cost, point-of-care screening for Sars-Cov-2.
Journal ArticleDOI
Actin-dependent localization of an RNA encoding a cell-fate determinant in yeast
TL;DR: It is proposed that Ash1 mRNA particles are transported into the daughter bud along actin filaments and are anchored at the distal tip of daughter buds in post-anaphase cells.
Journal ArticleDOI
Identification of an Asymmetrically Localized Determinant, Ash1p, Required for Lineage-Specific Transcription of the Yeast HO Gene
Anita Sil,Ira Herskowitz +1 more
TL;DR: Ash1p is a cell fate determinant that is asymmetrically localized to the daughter nucleus where it inhibits HO transcription and has a zinc finger motif related to those of GATA transcriptional regulators.
Journal ArticleDOI
Genetic Screens Identify Host Factors for SARS-CoV-2 and Common Cold Coronaviruses.
Ruofan Wang,Camille R. Simoneau,Jessie Kulsuptrakul,Mehdi Bouhaddou,Katherine A. Travisano,Jennifer M. Hayashi,Jennifer M. Hayashi,Jared Carlson-Stevermer,James Zengel,Christopher M. Richards,Parinaz Fozouni,Jennifer Oki,Lauren Rodriguez,Bastian Joehnk,Keith Walcott,Kevin Holden,Anita Sil,Jan E. Carette,Nevan J. Krogan,Melanie Ott,Melanie Ott,Andreas S. Puschnik +21 more
TL;DR: Pharmacological inhibition of phosphatidylinositol kinases and cholesterol homeostasis reduced replication of all three coronaviruses and offer important insights for the understanding of the coronavirus life cycle and the development of host-directed therapies.
Journal ArticleDOI
Amino acid substitutions in the structured domains of histones H3 and H4 partially relieve the requirement of the yeast SWI/SNF complex for transcription.
Warren D. Kruger,Craig L. Peterson,Anita Sil,C Coburn,G Arents,E N Moudrianakis,Ira Herskowitz +6 more
TL;DR: The ability of mutations affecting the structure of the histone octamer to relieve the need for SWI and SNF products supports the proposal that the SWI/SNF complex stimulates transcription by altering chromatin structure and can account for the apparent conservation of SWI or SNF proteins in eukaryotes other than yeast.