Carlos A. Suárez-Quian

TL;DR: Sertoli cells cultured on top of extracellular matrix components assume a phenotype and morphology more characteristic of the in vivo, differentiated cells, which induces a morphogenesis of the cells into cords, which closely resemble the organ from which the cells were dissociated and which provide an environment permissive for germ cell differentiation.

TL;DR: Evidence is provided that erythropoietin acts to stimulate neural progenitor cells and to prevent apoptosis in the embryonic brain and that induction of erythroid differentiation and its receptor by hypoxia may contribute to selective cell survival in the brain.

TL;DR: The presence of AR in Lydig cells is consistent with the hypothesis that androgens modify Leydig cell activity in an autocrine fashion, and AR immunoreactivity in both Sertoli and peritubular myoid cells suggests their involvement in the androgenic control of spermatogenesis.

TL;DR: It is concluded that FKBP52 is an AR folding factor that has critically important physiological roles in some male reproductive tissues and can enhance AR-mediated transactivation.

TL;DR: The distribution of the androgen receptor (AR) in archival human testes was determined immunocytochemically using an affinity-purified peptide-specific rabbit antibody, PG21, and employing a modified biotin-streptavidin-immunoperoxidase method that incorporated a biotin amplification step.