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Carlos Cordon-Cardo

Researcher at Icahn School of Medicine at Mount Sinai

Publications -  620
Citations -  91832

Carlos Cordon-Cardo is an academic researcher from Icahn School of Medicine at Mount Sinai. The author has contributed to research in topics: Cancer & Prostate cancer. The author has an hindex of 144, co-authored 589 publications receiving 84862 citations. Previous affiliations of Carlos Cordon-Cardo include The Rogosin Institute & Erasmus University Rotterdam.

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H-RAS mutation is a key molecular feature of pediatric urothelial bladder cancer. A detailed report of three cases.

TL;DR: Pediatric tumors are characterized by a consistent H-RAS mutation status, whereas FGFR3 and p53 pathways are not involved in this tumor initiation, which may explain the few recurrences seen in this population.
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Implementation of a Precision Pathology Program Focused on Oncology-Based Prognostic and Predictive Outcomes

TL;DR: The following review article will focus on the evolution of predictive pathology from a subjective, ‘opinion-based’ approach to a quantitative science.
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Broad SARS-CoV-2 cell tropism and immunopathology in lung tissues from fatal COVID-19

TL;DR: In lung tissues from severely affected COVID-19 patients, there is evidence for broad SARS-CoV-2 cell tropisms, activation of immune cells, and clearance of immunosuppressive cells, which could contribute to severe tissue damage, thromboemboli, excess inflammation and compromised adaptive immune responses.
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Application of new in situ hybridization probes for Ku70 and Ku80 in tissue microarrays of paraffin-embedded malignant melanomas: correlation with immunohistochemical analysis

TL;DR: The in situ hybridization assay for the detection of Ku70 and Ku80 expression used in this study is also suitable for tissue microarray analysis of paraffin-embedded melanoma samples.
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Loss of Ku70/Ku80 expression occurs more frequently in hereditary than in sporadic colorectal tumors. Tissue microarray study

TL;DR: It is concluded that expression of Ku70/Ku80 genes is down-regulated in colorectal carcinoma and that defects of these genes are more frequently observed in hereditary than in sporadic tumors.