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Carlos Cordon-Cardo
Researcher at Icahn School of Medicine at Mount Sinai
Publications - 620
Citations - 91832
Carlos Cordon-Cardo is an academic researcher from Icahn School of Medicine at Mount Sinai. The author has contributed to research in topics: Cancer & Prostate cancer. The author has an hindex of 144, co-authored 589 publications receiving 84862 citations. Previous affiliations of Carlos Cordon-Cardo include The Rogosin Institute & Erasmus University Rotterdam.
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Journal ArticleDOI
Humoral response and PCR positivity in patients with COVID-19 in the New York City region, USA: an observational study.
Ania Wajnberg,Mayce Mansour,Emily Leven,Nicole M. Bouvier,Gopi Patel,Adolfo Firpo-Betancourt,Rao Mendu,Jeffrey S. Jhang,Suzanne Arinsburg,Melissa R. Gitman,Jane Houldsworth,Emilia Mia Sordillo,Alberto Paniz-Mondolfi,Ian Baine,Viviana Simon,Judith A. Aberg,Florian Krammer,David Reich,Carlos Cordon-Cardo +18 more
TL;DR: Analysis of a large cohort of convalescent serum donors in the New York City region suggests that most patients with mild COVID-19 seroconvert 4 weeks after illness, and raises questions about the use of PCR to clear positive individuals.
Journal ArticleDOI
Oncogenes in melanoma.
David Polsky,Carlos Cordon-Cardo +1 more
TL;DR: A general overview of the mechanisms of oncogene activation and the functions of Oncogenes is provided, and oncogenic events in melanoma are reviewed.
Journal Article
Stimulation of T lymphocyte proliferation by monoclonal antibodies against GD3 ganglioside.
Karl Welte,G Miller,P B Chapman,H Yuasa,E Natoli,J E Kunicka,Carlos Cordon-Cardo,C Buhrer,Lloyd J. Old,Alan N. Houghton +9 more
TL;DR: It is demonstrated that binding to GD3 on the surface of T cells can elicit signals for T cell proliferation and could be augmented by exogenous interleukin 2 and phytohemagglutinin.
Journal ArticleDOI
Alveolar rhabdomyosarcoma: Is the cell of origin a mesenchymal stem cell?
TL;DR: A potential role for mesenchymal stem cells as the cell of origin of ARMS is explored, and the possibility that PAX-FKHR fusion genes may commit these cells to a myogenic lineage while inhibiting terminal differentiation, thus contributing to ARMS formation is explored.
Journal ArticleDOI
3-Phosphoinositide-Dependent Kinase 1 Potentiates Upstream Lesions on the Phosphatidylinositol 3-Kinase Pathway in Breast Carcinoma
Matthew Maurer,Tao Su,Lao H. Saal,Susan Koujak,Benjamin D. Hopkins,Christina R. Barkley,Jiaping Wu,Subhadra V. Nandula,Bhaskar Dutta,Yuli Xie,Y. Rebecca Chin,Da In Kim,Jennifer S. Ferris,Sofia K. Gruvberger-Saal,Mervi Laakso,Xiaomei Wang,Lorenzo Memeo,Albert Rojtman,Tulio Matos,Jennifer S. Yu,Carlos Cordon-Cardo,Jorma Isola,Mary Beth Terry,Alex Toker,Gordon B. Mills,Jean J. Zhao,Vundavalli V. Murty,Hanina Hibshoosh,Ramon Parsons +28 more
TL;DR: It is argued that PDK1 overexpression and increased PDPK1 copy number are common occurrences in cancer that potentiate the oncogenic effect of upstream lesions on the PI3K pathway.