C
Carmine V. Oddis
Researcher at Vanderbilt University
Publications - 3
Citations - 1247
Carmine V. Oddis is an academic researcher from Vanderbilt University. The author has contributed to research in topics: Ca2+/calmodulin-dependent protein kinase & Transgene. The author has an hindex of 3, co-authored 3 publications receiving 1140 citations. Previous affiliations of Carmine V. Oddis include Veterans Health Administration.
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Journal ArticleDOI
A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation
Jeffrey R. Erickson,Mei Ling A. Joiner,Xiaoqun Guan,William Kutschke,Jinying Yang,Carmine V. Oddis,Ryan K. Bartlett,John S. Lowe,Susan E. O'Donnell,Nukhet Aykin-Burns,Matthew C. Zimmerman,Kathy Zimmerman,Amy-Joan L. Ham,Robert M. Weiss,Douglas R. Spitz,Madeline A. Shea,Roger J. Colbran,Peter J. Mohler,Mark E. Anderson +18 more
TL;DR: It is shown that oxidation of paired regulatory domain methionine residues sustains CaMKII activity in the absence of Ca2+/CaM and highlights the critical importance of oxidation-dependent CaMK II activation to AngII and ischemic myocardial apoptosis.
Journal ArticleDOI
Calmodulin kinase II inhibition protects against myocardial cell apoptosis in vivo
Yingbo Yang,Weizhong Zhu,Mei Ling A. Joiner,Rong Zhang,Carmine V. Oddis,Yue Hou,Jinying Yang,Edward Price,Linda A. Gleaves,Mesut Eren,Gemin Ni,Douglas E. Vaughan,Rui-Ping Xiao,Mark E. Anderson +13 more
TL;DR: Findings indicate CaMKII is proapoptotic in vivo and suggest that regulation of SR Ca(2+) content by PLN contributes to the antiapoptosis mechanism of CaMK II inhibition.
Journal ArticleDOI
Death, Cardiac Dysfunction, and Arrhythmias Are Increased by Calmodulin Kinase II in Calcineurin Cardiomyopathy
Michelle S.C. Khoo,Jingdong Li,Jingdong Li,Madhu V. Singh,Yingbo Yang,Prince J. Kannankeril,Yuejin Wu,Chad E. Grueter,Xiaoqun Guan,Carmine V. Oddis,Rong Zhang,Lisa A. Mendes,Gemin Ni,Ernest C. Madu,Jinying Yang,Martha A. Bass,Rey J. Gomez,Brian E. Wadzinski,Eric N. Olson,Roger J. Colbran,Mark E. Anderson +20 more
TL;DR: CaMKII is a downstream signal in CAN cardiomyopathy, and increased CaMKII activity contributes to cardiac dysfunction, arrhythmia susceptibility, and longevity during CAN overexpression.