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Donald V. Lightner

Researcher at University of Arizona

Publications -  205
Citations -  13332

Donald V. Lightner is an academic researcher from University of Arizona. The author has contributed to research in topics: Shrimp & Infectious hypodermal and hematopoietic necrosis. The author has an hindex of 58, co-authored 205 publications receiving 12079 citations.

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A survey of cultured penaeid shrimp in Taiwan for viral and other important diseases

TL;DR: A survey of disease problems adversely affecting cultured shrimp at farms in Southern Taiwan was conducted in March of 1986, and findings indicate that MBV is a common infectious agent in cultured stocks of these species in Taiwan.
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Taura syndrome of penaeid shrimp: cloning of viral genome fragments and development of specific gene probes.

TL;DR: The ssRNA genome extracted from purified Taura Syndrome Virus was transcribed into double-stranded, blunt-ended cDNA and was used to construct cDNA libraries either in pUC 18 or in pBluescript II KS-vectors and the specificity of the 2 probes was confirmed by in situ hybridization on histological sections of TS diseased shrimps.
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qPCR assay for detecting and quantifying a virulence plasmid in acute hepatopancreatic necrosis disease (AHPND) due to pathogenic Vibrio parahaemolyticus

TL;DR: This qPCR assay is specific with high sensitivity (10 copies of virulence plasmid) and can be used to detect AHPND-pathogenic V. parahaemolyticus in shrimp and water samples.
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The geographic distribution of Taura Syndrome Virus (TSV) in the Americas : determination by histopathology and in situ hybridization using TSV-specific cDNA probes

TL;DR: The findings further strengthen the existing evidence that TS has a viral, not a toxic, etiology and indicate that either a single TSV strain, or very similar strains of the same virus, are responsible for the TSV panzootic that has been expanding in the Americas since 1992.
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Quantification of white spot syndrome virus DNA through a competitive polymerase chain reaction

TL;DR: The number of WSSV genomes in both hemolymph and tissues corresponded to the severity of infection determined by histological evaluation as well as to the concentration of the internal standard.