E
Erica Travaglino
Researcher at Humanitas University
Publications - 82
Citations - 5974
Erica Travaglino is an academic researcher from Humanitas University. The author has contributed to research in topics: Myelodysplastic syndromes & Myeloid. The author has an hindex of 25, co-authored 76 publications receiving 5041 citations. Previous affiliations of Erica Travaglino include University of Pavia.
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Journal ArticleDOI
Somatic SF3B1 Mutation in Myelodysplasia with Ring Sideroblasts
Elli Papaemmanuil,Mario Cazzola,Jacqueline Boultwood,Luca Malcovati,Paresh Vyas,David T. Bowen,Andrea Pellagatti,J. S. Wainscoat,Eva Hellström-Lindberg,Carlo Gambacorti-Passerini,Anna L. Godfrey,I. Rapado,Ana Cvejic,Ana Cvejic,Richard Rance,C. McGee,Peter R. Ellis,Laura Mudie,Phil Stephens,Stuart McLaren,Charles E. Massie,Patrick S. Tarpey,Ignacio Varela,Serena Nik-Zainal,Helen Davies,Adam Shlien,David T. Jones,Keiran Raine,Jonathon Hinton,Adam Butler,Jon W. Teague,E J Baxter,Joannah Score,Anna Gallì,M.G. Della Porta,Erica Travaglino,Michael J. Groves,Sudhir Tauro,Nikhil C. Munshi,Nikhil C. Munshi,KC Anderson,Adel K. El-Naggar,Andrej Fischer,Andrej Fischer,Ville Mustonen,Alan J. Warren,Nicholas C.P. Cross,Anthony R. Green,P A Futreal,Michael R. Stratton,Peter J. Campbell,Peter J. Campbell +51 more
TL;DR: Mutations in SF3B1 implicate abnormalities of messenger RNA splicing in the pathogenesis of myelodysplastic syndromes and were associated with down-regulation of key gene networks, including core mitochondrial pathways.
Journal ArticleDOI
Prognostic Factors and Life Expectancy in Myelodysplastic Syndromes Classified According to WHO Criteria: A Basis for Clinical Decision Making
Luca Malcovati,Matteo G. Della Porta,Cristiana Pascutto,Rosangela Invernizzi,Marina Boni,Erica Travaglino,Francesco Passamonti,Luca Arcaini,Margherita Maffioli,Paolo Bernasconi,Mario Lazzarino,Mario Cazzola +11 more
TL;DR: Data show that the WHO classification of MDSs has a relevant prognostic value, and this classification, along with cytogenetics, might be useful in decisions regarding transplantation.
Journal ArticleDOI
Clinical significance of SF3B1 mutations in myelodysplastic syndromes and myelodysplastic/myeloproliferative neoplasms
Luca Malcovati,Elli Papaemmanuil,David T. Bowen,Jacqueline Boultwood,Matteo G. Della Porta,Cristiana Pascutto,Erica Travaglino,Michael J. Groves,Anna L. Godfrey,Ilaria Ambaglio,Anna Gallì,Matteo Claudio Da Via,Simona Conte,Sudhir Tauro,Norene Keenan,Ann Hyslop,Jonathan Hinton,Laura Mudie,James S. Wainscoat,P. Andrew Futreal,Michael R. Stratton,Peter J. Campbell,Eva Hellström-Lindberg,Mario Cazzola +23 more
TL;DR: It is found that SF3B1 mutations are independent predictors of favorable clinical outcome, and their incorporation into stratification systems might improve risk assessment in MDS.
Journal ArticleDOI
Clinical significance of somatic mutation in unexplained blood cytopenia
Luca Malcovati,Anna Gallì,Erica Travaglino,Ilaria Ambaglio,Ettore Rizzo,Elisabetta Molteni,Chiara Elena,Virginia Valeria Ferretti,Silvia Catricalà,Elisa Bono,Gabriele Todisco,Antonio Bianchessi,Elisa Rumi,Silvia Zibellini,Daniela Pietra,Emanuela Boveri,Clara Camaschella,Daniela Toniolo,Elli Papaemmanuil,Seishi Ogawa,Mario Cazzola +20 more
TL;DR: Mutation analysis on peripheral blood granulocytes may significantly improve the current diagnostic approach to unexplained cytopenia and more generally the diagnostic accuracy of myeloid neoplasms.
Journal ArticleDOI
SF3B1 mutation identifies a distinct subset of myelodysplastic syndrome with ring sideroblasts.
Luca Malcovati,Mohsen Karimi,Elli Papaemmanuil,Ilaria Ambaglio,Martin Jädersten,Monika Jansson,Chiara Elena,Anna Gallì,Gunilla Walldin,Matteo G. Della Porta,Klas Raaschou-Jensen,Erica Travaglino,Klaus Kallenbach,Daniela Pietra,Viktor Ljungström,Simona Conte,Emanuela Boveri,Rosangela Invernizzi,Richard Rosenquist,Peter J. Campbell,Mario Cazzola,Eva Hellstrom Lindberg +21 more
TL;DR: SF3B1 mutation identifies a distinct MDS subtype that is unlikely to develop detrimental subclonal mutations and is characterized by indolent clinical course and favorable outcome.