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Gilles Dietrich

Researcher at University of Toulouse

Publications -  77
Citations -  3188

Gilles Dietrich is an academic researcher from University of Toulouse. The author has contributed to research in topics: Autoantibody & Antibody. The author has an hindex of 30, co-authored 71 publications receiving 2798 citations. Previous affiliations of Gilles Dietrich include Centre national de la recherche scientifique & Université catholique de Louvain.

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Journal ArticleDOI

Inflammation and Gut-Brain Axis During Type 2 Diabetes: Focus on the Crosstalk Between Intestinal Immune Cells and Enteric Nervous System.

Arnaud Bessac, +6 more
- 10 Oct 2018 - 
TL;DR: The role of the crosstalk between intestinal immune cells and ENS neurons in the control of glycemia is focused on and its potential contribution and role on the immune and neuronal systems are discussed.
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Delta opioid receptors mediate chemotaxis in bone marrow-derived dendritic cells.

Alan Bénard, +10 more
- 15 Jun 2008 - 
TL;DR: It was found that the delta opioid receptor (DOR) mRNA was expressed at low level in bone marrow-derived immature DCs and up-regulated upon DC maturation and triggered DC chemotaxis in vitro, suggesting that the opioid chemotactic activity decreases as the concentration of the chemokines increases.
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Evidence for a restricted idiotypic and epitopic specificity of anti-thyroglobulin autoantibodies in patients with autoimmune thyroiditis.

Gilles Dietrich, +3 more
- 01 Mar 1991 - 
TL;DR: The present study demonstrates that the expression of the T44 Id is strongly associated with the recognition by anti‐TG autoantibodies of a specific epitopic cluster on human TG, suggesting that qualitative rather than quantitative criteria should be used to identify pathological autoantsibodies.
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Antiidiotypic suppression of autoantibodies with normal polyspecific immunoglobulins.

F. Rossi, +2 more
- 01 Jan 1989 - 
TL;DR: The presence of antiidiotypes against autoantibodies in pooled normal IgG supports the concept of a functional idiotypic network regulating autoimmune responses in man.
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The cGMP/protein kinase G pathway contributes to dihydropyridine-sensitive calcium response and cytokine production in TH2 lymphocytes.

Bruno Gomes, +10 more
- 05 May 2006 - 
TL;DR: Data indicate that, in Th2 cells, cGMP is produced upon TCR engagement and activates PKG, which controls DHP-sensitive calcium inflow and Th2 cytokine production.