J
Jeremy D. Walston
Researcher at Johns Hopkins University
Publications - 337
Citations - 48109
Jeremy D. Walston is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Population & Medicine. The author has an hindex of 80, co-authored 302 publications receiving 39548 citations. Previous affiliations of Jeremy D. Walston include Johns Hopkins University School of Medicine & University of Baltimore.
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Journal ArticleDOI
β3-adrenoceptor gene variant in obesity and insulin resistance
Journal ArticleDOI
Development and validation of a falls-grading scale
Marcela Davalos-Bichara,Frank R. Lin,John P. Carey,Jeremy D. Walston,Jennifer E. Fairman,Michael C. Schubert,Jeremy S. Barron,Jennifer M. Hughes,Jennifer L. Millar,Anne Spar,Kristy L. Weber,Howard S. Ying,Kathleen M. Zackowski,David S. Zee,Yuri Agrawal +14 more
TL;DR: A scale to grade near-fall and fall events on the basis of their severity represented by the use of health care resources, with the goal of standardizing fall reporting in the clinical and research settings is developed and validated.
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Hepcidin‐dependent and hepcidin‐independent regulation of erythropoiesis in a mouse model of anemia of chronic inflammation
Jacqueline M. Langdon,Saiah C. Yates,Laurette K. Femnou,Bryan J. McCranor,Chris Cheadle,Qian Li Xue,Qian Li Xue,Sophie Vaulont,Curt I. Civin,Jeremy D. Walston,Cindy N. Roy +10 more
TL;DR: Differences in erythrocyte parameters suggest anemia in many inflammatory states may not be fully explained by hepcidin‐mediated iron sequestration, and chronic anemia associated with inflammation may benefit from interventions protecting ery Throcyte number in addition to anti‐hePCidin interventions aimed at enhancing iron availability.
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Critical Transition in Tissue Homeostasis Accompanies Murine Lung Senescence
Carla L. Calvi,Megan Podowski,Franco R. D'Alessio,Shana Metzger,Kaori Misono,Hataya Poonyagariyagorn,Armando Lopez-Mercado,Therese Ku,Thomas Lauer,Christopher Cheadle,C. Conover Talbot,Chunfa Jie,Sharon A. McGrath-Morrow,Landon S. King,Jeremy D. Walston,Enid Neptune +15 more
TL;DR: It is established that a tissue-specific aging program is evident during a presenescent interval which involves early oxidative stress, cell death and elastase activation, followed by B lymphocyte and macrophage expansion/activation, which heralds the progression to overt airspace enlargement in the aged lung.
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Evidence for dying‐back axonal degeneration in age‐associated skeletal muscle decline
TL;DR: Age‐associated muscle strength decline is a major contributing factor to increased late‐life functional decline and comorbidity, and is strongly associated with early mortality.