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Jeremy D. Walston

Researcher at Johns Hopkins University

Publications -  337
Citations -  48109

Jeremy D. Walston is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Population & Medicine. The author has an hindex of 80, co-authored 302 publications receiving 39548 citations. Previous affiliations of Jeremy D. Walston include Johns Hopkins University School of Medicine & University of Baltimore.

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Knockout of Angiotensin AT2 receptors accelerates healing but impairs quality.

TL;DR: The results suggest that in the absence of AT2R, wound healing rate is accelerated, but yielded worse skin quality, and elucidating the contribution of both of the angiotensin receptors may help fine tune future intervention aimed at wound repair in older individuals.
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Aged interleukin-10tm1Cgn chronically inflamed mice have substantially reduced fat mass, metabolic rate, and adipokines

TL;DR: Fat mass is decreased with age in IL 10tm mice compared to controls and NMR based body composition analysis and dissection weights suggest that frailty observed in this mouse model of chronic inflammation may in part be driven by alterations in fat mass, hormone secretion and energy metabolism.
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Interferon-γ Potentiates α-Synuclein-induced Neurotoxicity Linked to Toll-like Receptors 2 and 3 and Tumor Necrosis Factor-α in Murine Astrocytes.

TL;DR: Interferon (IFN)-γ potentiates α-synuclein (A53T or wild-type)-induced innate immunity, involving expressions of TLR2, TLR3, NF-κB, and TNF-α, other than IL-1β, suggesting that involvement of IFN-γ in α- synuclein's innate immunity may be required for initiation and maintenance of glial activation, a novel neurotoxic mechanism underlying pathogenesis of neurodegenerative