J
John B. Winer
Researcher at Queen Elizabeth Hospital Birmingham
Publications - 10
Citations - 2069
John B. Winer is an academic researcher from Queen Elizabeth Hospital Birmingham. The author has contributed to research in topics: Sensory nerve & Essential hypertension. The author has an hindex of 8, co-authored 10 publications receiving 1864 citations. Previous affiliations of John B. Winer include University of Birmingham.
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Journal ArticleDOI
The American College of Rheumatology nomenclature and case definitions for neuropsychiatric lupus syndromes
Matthew H. Liang,Michael Corzillius,Sang Cheol Bae,Robert A. Lew,Paul R. Fortin,Caroline Gordon,David A. Isenberg,Graciela S. Alarcón,Karin V. Straaton,Judah A. Denburg,Susan D. Denburg,John M. Esdaile,Bonnie I. Glanz,Elizabeth W. Karlson,Shahram Khoshbin,Malcolm P. Rogers,Peter H. Schur,John G. Hanly,Elizabeth Kozora,Sterling G. West,Robert G. Lahita,Michael D. Lockshin,Joseph M. McCune,Patricia M. Moore,Michelle Petri,W. Neal Roberts,Jorge Sánchez-Guerrero,Martin Veilleux,Robin L. Brey,Wayne D. Cornblath,Christopher M. Filley,John D. Fisk,Pontus Harten,Elaine M. Hay,Grant L. Iverson,Steven R. Levine,Elizabeth Waterhouse,Daniel J. Wallace,John B. Winer +38 more
TL;DR: The American College of Rheumatology Nomenclature for NPSLE provides case definitions for 19 neuropsychiatric syndromes seen in SLE, with reporting standards and recommendations for laboratory and imaging tests.
Journal ArticleDOI
Sensory detection thresholds are modulated across the cardiac cycle: evidence that cutaneous sensibility is greatest for systolic stimulation.
TL;DR: The finding that cutaneous sensibility was greater when stimulated during systole than diastole may be accounted for by a modified afferent feedback hypothesis.
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γδ T cells infiltrating sensory nerve biopsies from patients with inflammatory neuropathy
TL;DR: The presence of T cells of a γδ T cell receptor phenotype in nerve biopsy specimens from patients with inflammatory neuropathy is consistent with a possible pathogenetic role of a cellular immune response against non-protein antigens such as gangliosides.
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Guillain–Barré syndrome: Clinical variants and their pathogenesis
TL;DR: Numerous clinical subtypes of Guillain-Barré syndrome have been described over the century since the original description of the syndrome and their immunological pathogenesis is reviewed.
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Cutaneous sensibility and peripheral nerve function in patients with unmedicated essential hypertension.
TL;DR: It is suggested that hypertension may reduce the number of active sensory nerve fibers without affecting myelination and subclinical axonal neuropathy of sensory afferents may help account for perceptual deficits that characterize hypertension.