J
Joseph M. Pilewski
Researcher at University of Pittsburgh
Publications - 322
Citations - 17263
Joseph M. Pilewski is an academic researcher from University of Pittsburgh. The author has contributed to research in topics: Lung transplantation & Cystic fibrosis. The author has an hindex of 65, co-authored 285 publications receiving 15142 citations. Previous affiliations of Joseph M. Pilewski include Boston Children's Hospital & University of Pennsylvania.
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Journal ArticleDOI
Post-transplant Colonization With Non-Aspergillus Molds and Risk of Development of Invasive Fungal Disease in Lung Transplant Recipients
Fernanda P. Silveira,Eun J. Kwak,David L. Paterson,David L. Paterson,Joseph M. Pilewski,Kenneth R. McCurry,Shahid Husain +6 more
TL;DR: Isolation of non-Aspergillus molds in the BAL of LTx recipients may not be associated with the development of invasive disease, irrespective of anti-fungal prophylaxis.
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Impact of a Mobile Health Intervention on Long-term Nonadherence After Lung Transplantation: Follow-up After a Randomized Controlled Trial.
Emily M Geramita,Annette J. DeVito Dabbs,Andrea DiMartini,Joseph M. Pilewski,Galen E. Switzer,Donna M. Posluszny,Larissa Myaskovsky,Mary Amanda Dew +7 more
TL;DR: Pocket PATH did not have sustained impact on most areas of the regimen, although the Pocket PATH group showed lower risk of nonadherence to lifestyle requirements (diet/exercise) than the usual care group (P < 0.05).
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Syndecan-2 is a novel target of insulin-like growth factor binding protein-3 and is over-expressed in fibrosis.
Ximena D. Ruiz,Logan R Mlakar,Yukie Yamaguchi,Yunyun Su,Adriana T. Larregina,Joseph M. Pilewski,Carol Feghali-Bostwick +6 more
TL;DR: Increased SDC2 expression is due, at least in part, to the activity of two pro-fibrotic factors, TGFβ and IGFBP-3, which is over-expressed in fibrotic tissues.
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Finally, mice with CF lung disease
TL;DR: The development of lung disease in these mice should provide insights into a disease that has long been lacking an animal model of cystic fibrosis.
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Delayed chest closure after lung transplantation: Techniques, outcomes, and strategies
Norihisa Shigemura,Yucel Orhan,Jay K. Bhama,Jonathan D'Cunha,Diana Zaldonis,Joseph M. Pilewski,James D. Luketich,Christian Bermudez +7 more
TL;DR: DCC contributed to significantly decreased incidence of severe PGD, leading to an improved post-transplant survival and functional status as compared with primary closure, and should not be considered a sub-optimal option after lung transplantation.