K
Kazuya Zeki
Researcher at University of Occupational and Environmental Health Japan
Publications - 24
Citations - 785
Kazuya Zeki is an academic researcher from University of Occupational and Environmental Health Japan. The author has contributed to research in topics: Cell culture & Thyroid. The author has an hindex of 13, co-authored 24 publications receiving 767 citations.
Papers
More filters
Journal ArticleDOI
Expression of the neoplastic phenotype by human thyroid carcinoma cell lines requires NFkappaB p65 protein expression.
Roberta Visconti,Janete Cerutti,Sabrina Battista,Monica Fedele,Francesco Trapasso,Kazuya Zeki,Maria Pia Miano,Filomena de Nigris,Laura Casalino,Francesco Curcio,Massimo Santoro,Alfredo Fusco +11 more
TL;DR: The results indicate that activation of the NFκB complex by overexpression of p65 plays a critical role in the process of thyroid cell transformation.
Journal ArticleDOI
Interleukin-4 as a potent inhibitor of bone resorption
Kenichi Watanabe,Yoshiya Tanaka,Isao Morimoto,Katsuya Yahata,Kazuya Zeki,Takashi Fujihira,Uki Yamashita,Sumiya Eto +7 more
TL;DR: Results suggest that IL-4 may play an important role on the inhibitory regulation of bone resorption.
Journal ArticleDOI
Sensitivity difference to the suppressive effect of prostaglandin E2 among mouse strains: a possible mechanism to polarize Th2 type response in BALB/c mice.
TL;DR: It is proposed that the modulation of P GE2 is important for Th1 activation via IFN-γ and IL-12p70 production in vitro and in vivo and that PGE2 is one of the pivotal factors in the Th2-dominant immune response in BALB/c mice.
Journal ArticleDOI
Prostaglandin E2 Up-Regulates Macrophage-Derived Chemokine Production but Suppresses IFN-Inducible Protein-10 Production by APC
TL;DR: Results indicate that P GE2 regulates Th1/Th2-related chemokine production by B cells, macrophages, and dendritic cells, and that this is a new function of PGE2 for the regulation of Th2 immune responses at the induction and activation stages.
Journal ArticleDOI
Parathyroid hormone-related peptide-(1-34) [PTHrP-(1-34)] induces vasopressin release from the rat supraoptic nucleus in vitro through a novel receptor distinct from a type I or type II PTH/PTHrP receptor.
Shigeki Yamamoto,Isao Morimoto,Nobuyuki Yanagihara,Kazuya Zeki,Takashi Fujihira,Futoshi Izumi,Hiroshi Yamashita,Sumiya Eto +7 more
TL;DR: Application of PTHrP-(1–34) to SON slices caused an increase in AVP release in a concentration-dependent manner and dose-dependently increased the levels of cAMP in the SON.