L
Laura Giribaldi
Researcher at University of Buenos Aires
Publications - 3
Citations - 213
Laura Giribaldi is an academic researcher from University of Buenos Aires. The author has contributed to research in topics: Galectin-1 & Cancer. The author has an hindex of 3, co-authored 3 publications receiving 187 citations. Previous affiliations of Laura Giribaldi include Facultad de Ciencias Exactas y Naturales & National Scientific and Technical Research Council.
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Journal ArticleDOI
A unique galectin signature in human prostate cancer progression suggests galectin-1 as a key target for treatment of advanced disease
Diego Jose Laderach,Lucas Daniel Gentilini,Laura Giribaldi,Víctor M. Cárdenas Delgado,Lorena G. Nugnes,Diego O. Croci,Nader Al Nakouzi,Paula Alejandra Sacca,Gabriel Casas,Osvaldo Mazza,Margaret A. Shipp,Elba Vazquez,Anne Chauchereau,Jeffery L. Kutok,Scott J. Rodig,María T. Elola,Daniel Compagno,Gabriel A. Rabinovich +17 more
TL;DR: A dynamically regulated "galectin-specific signature" that accompanies disease evolution in prostate cancer is identified and highlighted, highlighting a major role for Gal-1 as a tractable target for antiangiogenic therapy in advanced stages of the disease.
Journal ArticleDOI
Galectin-1 confers immune privilege to human trophoblast: implications in recurrent fetal loss.
Rosanna Ramhorst,Laura Giribaldi,Laura Giribaldi,Laura Fraccaroli,Marta A. Toscano,Juan C. Stupirski,M.D. Romero,Edith Sandra Durand,Natalia Rubinstein,Astrid Blaschitz,Peter Sedlmayr,Susana Genti-Raimondi,Leonardo Fainboim,Gabriel A. Rabinovich,Gabriel A. Rabinovich +14 more
TL;DR: Exogenous Gal1 confers immune privilege to human trophoblast cells by triggering a broad tolerogenic program with potential implications in threatened pregnancies.
Journal ArticleDOI
Hexachlorobenzene induces deregulation of cellular growth in rat liver.
Laura Giribaldi,Florencia Chiappini,Carolina Pontillo,Andrea Randi,Diana Kleiman de Pisarev,Laura Alvarez +5 more
TL;DR: It is reported for the first time that HCB induces loss of the homeostatic balance between cell growth and cell death in rat liver, by mechanisms involving signals emanating from death receptors, and the mitochondrial pathway.