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Lei Wei

Researcher at Wuhan University

Publications -  320
Citations -  13830

Lei Wei is an academic researcher from Wuhan University. The author has contributed to research in topics: Cartilage & Osteoarthritis. The author has an hindex of 57, co-authored 304 publications receiving 11840 citations. Previous affiliations of Lei Wei include Rhode Island Hospital & Brown University.

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Non-alcoholic fatty liver disease is associated with stroke severity and progression of brainstem infarctions.

Haiyan Li, +8 more
- 01 Mar 2018 - 
TL;DR: Non‐alcoholic fatty liver disease (NAFLD) is closely correlated to visceral obesity, dyslipidaemia, insulin resistance and type 2 diabetes mellitus and whether NAFLD is an independent risk factor in patients with BSIs is evaluated.
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Association of serum uric acid levels with the progression of Parkinson's disease in Chinese patients.

Sun Cc, +7 more
- 01 Feb 2012 - 
TL;DR: Lower serum UA levels may possess a higher risk of PD, which may be a potential useful biomarker to indicate the progression of PD.
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Protective Effect of Astragalus polysaccharides on Liver Injury Induced by Several Different Chemotherapeutics in Mice

Wen Liu, +7 more
- 06 Jan 2015 - 
TL;DR: DTX, CTX and EPI induce liver damage in a dose dependent manner, whereas APS exerted protective effects, and sections of liver tissue showed massive hepatotoxicity in CTXH group compared to the control group.
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IFN‐β suppresses experimental autoimmune neuritis in Lewis rats by inhibiting the migration of inflammatory cells into peripheral nervous tissue

Li-Ping Zou, +5 more
- 15 Apr 1999 - 
TL;DR: It is concluded that the observed suppression of EAN by rrIFN-beta is associated with a decrease in the migration of inflammatory cells into peripheral nervous tissue.
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Abnormal Mechanical Loading Induces Cartilage Degeneration by Accelerating Meniscus Hypertrophy and Mineralization After ACL Injuries In Vivo

Guoqing Du, +15 more
- 20 Jan 2016 - 
TL;DR: The study data suggest that the suppression of meniscus hypertrophy and calcification may decrease the risk of PTOA after ACL injuries.