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Lixin Wang

Researcher at Indiana University – Purdue University Indianapolis

Publications -  419
Citations -  13655

Lixin Wang is an academic researcher from Indiana University – Purdue University Indianapolis. The author has contributed to research in topics: Soil water & Biology. The author has an hindex of 53, co-authored 349 publications receiving 9822 citations. Previous affiliations of Lixin Wang include Hebei University of Science and Technology & Wayne State University.

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Recent Photosynthates Are the Primary Carbon Source for Soil Microbial Respiration in Subtropical Forests

TL;DR: In this paper , the authors found that recent photosynthates, not soil organic carbon (SOC), contributed 88% ± 12% of the carbon source fueling microbial respiration in tropical and subtropical forests.
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The moisture origin of dew: Insights from three sites with contrasting climatic conditions

TL;DR: In this article , the isotopic compositions of dew and precipitation were investigated at three sites with different climatic conditions (i.e., Gobabeb with extremely dry climate, Nice with Mediterranean climate and Indianapolis with humid continental climate).
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Stable isotope variations of dew under three different climates

TL;DR: In this article , the authors presented daily dew isotope dataset under three distinct climatic regions (i.e., Gobabeb in the central Namib Desert with desert climate, Nice in France with Mediterranean climate, and Indianapolis in central United States with humid continental climate) using a Triple Water Vapor Isotope Analyzer based on Off-Axis Integrated Cavity Output Spectroscopy technique.
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Quantitative clonal analysis reveals vast heterogeneity among fallopian tube cells for ovarian cancer-initiation and progression

TL;DR: This study investigated the capacity of fallopian tube cells in initiating serous ovarian cancer, utilizing a mouse genetic system that generates scattered GFP-labeled mutant cells and revealed that only a rare primitive subset enriched in the distal fallopian tubes is capable of clonal expansion whereas others stall immediately upon acquiring oncogenic mutations.