P
Pawel Fidzinski
Researcher at Humboldt University of Berlin
Publications - 37
Citations - 1552
Pawel Fidzinski is an academic researcher from Humboldt University of Berlin. The author has contributed to research in topics: Hippocampal formation & Long-term potentiation. The author has an hindex of 16, co-authored 37 publications receiving 1096 citations. Previous affiliations of Pawel Fidzinski include Free University of Berlin & Charité.
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Journal ArticleDOI
Dendritic action potentials and computation in human layer 2/3 cortical neurons.
Albert Gidon,Timothy A. Zolnik,Pawel Fidzinski,Felix Bolduan,Athanasia Papoutsi,Panayiota Poirazi,Martin Holtkamp,Imre Vida,Matthew E. Larkum,Matthew E. Larkum +9 more
TL;DR: In these neurons, a class of calcium-mediated dendritic action potentials whose waveform and effects on neuronal output have not been previously described are discovered, which enabled the dendrites of individual human neocortical pyramidal neurons to classify linearly nonseparable inputs—a computation conventionally thought to require multilayered networks.
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Human microglia regional heterogeneity and phenotypes determined by multiplexed single-cell mass cytometry.
Chotima Böttcher,Stephan Schlickeiser,Marjolein A. M. Sneeboer,Désirée Kunkel,Anniki Knop,Evdokia Paza,Pawel Fidzinski,Larissa Kraus,G. Snijders,René S. Kahn,Axel R. Schulz,Henrik E. Mei,Elly M. Hol,Elly M. Hol,Britta Siegmund,Rainer Glauben,Eike Jakob Spruth,Lot de Witte,Lot de Witte,Josef Priller +19 more
TL;DR: The phenotypic signature of hu MG was identified, which was distinct from peripheral myeloid cells but was comparable to fresh huMG, and microglia regional heterogeneity was identified.
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Ca2+-activated Cl− currents are dispensable for olfaction.
TL;DR: This work has identified Anoctamin2 (Ano2, also known as TMEM16B) as the ciliary Ca2+-activated Cl− channel of olfactory receptor neurons and disruption of Ano2 in mice virtually abolished Ca2 + Cl− currents in the MOE and VNO.
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Loss of GABAergic neurons in the subiculum and its functional implications in temporal lobe epilepsy
TL;DR: The data suggest that the vulnerability of subicular GABAergic interneurons causes an input-specific disturbance of the subicular inhibitory system in epileptic animals.
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The Late Endosomal ClC-6 Mediates Proton/Chloride Countertransport in Heterologous Plasma Membrane Expression
TL;DR: In this article, the green fluorescent protein (GFP) was fused to the N terminus of ClC-6, which increased its cell surface expression, allowing the first time that ClC6 is a Cl−/H+ antiporter.