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Peter C. Taylor

Researcher at University of Oxford

Publications -  397
Citations -  19020

Peter C. Taylor is an academic researcher from University of Oxford. The author has contributed to research in topics: Rheumatoid arthritis & Arthritis. The author has an hindex of 63, co-authored 359 publications receiving 16347 citations. Previous affiliations of Peter C. Taylor include HealthPartners & University of Cambridge.

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Journal ArticleDOI

Plasma Levels of Eicosapentaenoic Acid Are Associated with Anti-TNF Responsiveness in Rheumatoid Arthritis and Inhibit the Etanercept-driven Rise in Th17 Cell Differentiation in Vitro.

TL;DR: EPA status was associated with clinical improvements to anti-TNF therapy in vivo and prevented the effect of ETN on Th17 cells in vitro, suggesting that EPA supplementation might be a simple way to improve anti- TNF outcomes in patients with RA by suppressing Th17 frequencies.
Journal ArticleDOI

The down-regulation of IL1alpha and IL6, in monocytes exposed to extracorporeal photopheresis (ECP)-treated lymphocytes, is not dependent on lymphocyte phosphatidylserine externalization.

TL;DR: ECP‐treated lymphocytes can reduce the ability of LPS‐stimulated monocytes to produce some proinflammatory cytokines; however, this effect is not dependent on phosphatidylserine externalization.
Journal ArticleDOI

The ethics of ‘Trials within Cohorts’ (TwiCs): 2nd international symposium

Clare Relton, +56 more
- 20 Jul 2017 - 
TL;DR: It is possible to make a compelling case to ethics committees that TwiCs designs are appropriate and ethical, and some questions about the ethical requirements for content and timing of informed consent for a study using the TwiC design need to be decided on a case-by-case basis.
Book ChapterDOI

Impact of Comorbidity

TL;DR: It is proposed that the development of care plans encompassing a more holistic approach that incorporates comorbidities, rather than focusing on a single disease-orientated model, will greatly improve overall patient survival and quality of life in rheumatic diseases.
Journal ArticleDOI

Clonal analysis of immunoglobulin mRNA in rheumatoid arthritis synovium: characterization of expanded IgG3 populations.

TL;DR: The ratio of replacement/silent somatic mutations in these two families of clones suggests that the selective clonal expansion in the synovium of patients with rheumatoid arthritis is due to an antigen‐driven immune response.