P
Peter G. Gibson
Researcher at University of Newcastle
Publications - 774
Citations - 53254
Peter G. Gibson is an academic researcher from University of Newcastle. The author has contributed to research in topics: Asthma & Sputum. The author has an hindex of 103, co-authored 711 publications receiving 45722 citations. Previous affiliations of Peter G. Gibson include University of Sydney & National Health and Medical Research Council.
Papers
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Journal ArticleDOI
Gastro-oesophageal reflux treatment for asthma in adults and children.
TL;DR: There was no overall improvement in asthma following treatment for gastro-oesophageal reflux, and anti-reflux treatment did not consistently improve lung function, asthma symptoms, nocturnal asthma or the use of asthma medications.
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Airway inflammation is augmented by obesity and fatty acids in asthma
TL;DR: This was the first study to demonstrate an increase in neutrophilic airway inflammation in obese asthma, and this relationship was significant only in females with asthma; in males, saturated and monounsaturated fatty acids were important predictors of neutrophiles in asthma.
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A meta-analysis of adverse perinatal outcomes in women with asthma.
Vanessa E. Murphy,Jennifer A. Namazy,Heather Powell,Heather Powell,Michael Schatz,Christina D. Chambers,John Attia,Peter G. Gibson,Peter G. Gibson +8 more
TL;DR: Please cite this paper as: Murphy V, Namazy J, Powell H, Schatz M, Chambers C, Attia J, Gibson P. A meta‐analysis of adverse perinatal outcomes in women with asthma.
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Options for self-management education for adults with asthma.
Heather Powell,Peter G. Gibson +1 more
TL;DR: Optimal self-management allowing for optimisation of asthma control by adjustment of medications may be conducted by either self-adjustment with the aid of a written action plan or by regular medical review.
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Interleukin-8 Secretion and Neutrophil Recruitment Accompanies Induced Sputum Eosinophil Activation in Children with Acute Asthma
TL;DR: Airway inflammation is present during an acute exacerbation of asthma, and is characterized by infiltration and activation of both eosinophils and neutrophils, and the heterogeneity of airway inflammation in acute asthma may influence response to corticosteroid therapy.