P
Peter M. Sadow
Researcher at Harvard University
Publications - 231
Citations - 7025
Peter M. Sadow is an academic researcher from Harvard University. The author has contributed to research in topics: Thyroid & Thyroid carcinoma. The author has an hindex of 39, co-authored 201 publications receiving 5401 citations. Previous affiliations of Peter M. Sadow include Memorial Sloan Kettering Cancer Center & University of Pittsburgh.
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Journal ArticleDOI
Nomenclature Revision for Encapsulated Follicular Variant of Papillary Thyroid Carcinoma: A Paradigm Shift to Reduce Overtreatment of Indolent Tumors
Yuri E. Nikiforov,Raja R. Seethala,Giovanni Tallini,Zubair W. Baloch,Fulvio Basolo,Lester D.R. Thompson,Justine A. Barletta,Bruce M. Wenig,Abir Al Ghuzlan,Kennichi Kakudo,Thomas J. Giordano,Venancio Avancini Ferreira Alves,Elham Khanafshar,Sylvia L. Asa,Adel K. El-Naggar,William E. Gooding,Steven P. Hodak,Ricardo V. Lloyd,Guy Maytal,Ozgur Mete,Marina N. Nikiforova,Vania Nosé,Mauro Papotti,David N. Poller,Peter M. Sadow,Arthur S. Tischler,R. Michael Tuttle,Kathryn B. Wall,Virginia A. LiVolsi,Gregory W. Randolph,Ronald Ghossein +30 more
TL;DR: Thyroid tumors currently diagnosed as noninvasive EFVPTC have a very low risk of adverse outcome and should be termed NIFTP, and this reclassification will affect a large population of patients worldwide and result in a significant reduction in psychological and clinical consequences associated with the diagnosis of cancer.
Journal ArticleDOI
Performance of a Genomic Sequencing Classifier for the Preoperative Diagnosis of Cytologically Indeterminate Thyroid Nodules
Kepal N. Patel,Trevor E. Angell,Joshua E. Babiarz,Neil M. Barth,Thomas Blevins,Quan-Yang Duh,Ronald Ghossein,R. Mack Harrell,Jing Huang,Giulia C. Kennedy,Su Yeon Kim,Richard T. Kloos,Virginia A. LiVolsi,Gregory W. Randolph,Peter M. Sadow,Michael H. Shanik,Julie Ann Sosa,S. Thomas Traweek,P. Sean Walsh,Duncan Whitney,Michael W. Yeh,Paul W. Ladenson +21 more
TL;DR: Its 36% increase in specificity compared with the gene expression classifier potentially increases the number of patients with benign nodules who can safely avoid unnecessary diagnostic surgery.
Journal Article
Role of matrix metalloproteinases in failure to re-epithelialize after corneal injury.
M E Fini,William C. Parks,William B. Rinehart,M T Girard,M. Matsubara,James R. Cook,Judith A. West-Mays,Peter M. Sadow,Robert E. Burgeson,John J. Jeffrey,Michael B. Raizman,R. R. Krueger,James D. Zieske +12 more
TL;DR: Evidence is provided that over-expression of matrix metalloproteinases by resident corneal cells impedes re-epithelialization after some types of cornea injury, and treatment of thermally injured corneas with a synthetic inhibitor of matrix meetallop proteinases significantly improved basement membrane integrity.
Journal ArticleDOI
Renal cell carcinoma in tuberous sclerosis complex.
Ping Yang,Ping Yang,Kristine M. Cornejo,Peter M. Sadow,Liang Cheng,Mingsheng Wang,Yu Xiao,Zhong Jiang,Esther Oliva,Sergiusz Jozwiak,Robert L. Nussbaum,Adam S. Feldman,Elahna Paul,Elizabeth A. Thiele,Jane J. Yu,Elizabeth P. Henske,David J. Kwiatkowski,Robert H. Young,Chin-Lee Wu +18 more
TL;DR: In this article, the authors reported a sizable cohort of renal tumors seen in TSC and were able to identify them as different morphotypes, which may help to expand the morphologic spectrum of TSC-associated RCC.
Journal ArticleDOI
Widespread Chromosomal Losses and Mitochondrial DNA Alterations as Genetic Drivers in Hürthle Cell Carcinoma
Raj K. Gopal,K Kübler,K Kübler,Sarah E. Calvo,Sarah E. Calvo,Sarah E. Calvo,Paz Polak,Dimitri Livitz,Daniel Rosebrock,Peter M. Sadow,Braidie Campbell,Samuel E. Donovan,Salma Amin,Benjamin J. Gigliotti,Zenon Grabarek,Zenon Grabarek,Zenon Grabarek,Julian M. Hess,Chip Stewart,Lior Z. Braunstein,Peter F. Arndt,Scott Mordecai,Angela R. Shih,Frances L Chaves,Tiannan Zhan,Carrie C. Lubitz,Jiwoong Kim,A. John Iafrate,Lori J. Wirth,Sareh Parangi,Ignaty Leshchiner,Gilbert H. Daniels,Vamsi K. Mootha,Dora Dias-Santagata,Gad Getz,David G. McFadden +35 more
TL;DR: In this paper, the authors performed whole-exome sequencing on a cohort of primary, recurrent, and metastatic tumors, and identified recurrent mutations in DAXX, TP53, NRAS, NF1, CDKN1A, ARHGAP35, and the TERT promoter.