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Philip A. Gregory

Researcher at University of South Australia

Publications -  60
Citations -  10594

Philip A. Gregory is an academic researcher from University of South Australia. The author has contributed to research in topics: Epithelial–mesenchymal transition & microRNA. The author has an hindex of 31, co-authored 55 publications receiving 9462 citations. Previous affiliations of Philip A. Gregory include Flinders Medical Centre & Flinders University.

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The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1.

TL;DR: It is found that all five members of the microRNA-200 family were markedly downregulated in cells that had undergone EMT in response to transforming growth factor (TGF)-β or to ectopic expression of the protein tyrosine phosphatase Pez, suggesting that downregulation of themicroRNAs may be an important step in tumour progression.
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The RNA binding protein quaking regulates formation of circRNAs.

TL;DR: It is shown that hundreds of circRNAs are regulated during human epithelial-mesenchymal transition (EMT) and that the production of over one-third of abundant circ RNAs is dynamically regulated by the alternative splicing factor, Quaking (QKI), which itself is regulated during EMT.
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A Double-Negative Feedback Loop between ZEB1-SIP1 and the microRNA-200 Family Regulates Epithelial-Mesenchymal Transition

TL;DR: A double-negative feedback loop controlling ZEB1-SIP1 and miR-200 family expression that regulates cellular phenotype is established and has direct relevance to the role of these factors in tumor progression.
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MicroRNAs as regulators of epithelial-mesenchymal transition

TL;DR: A review of recent findings and their implications in both developmental EMT and tumor progression describes the molecular reprogramming and phenotypic changes involved in the conversion of polarised immotile epithelial cells to motile mesenchymal cells.