S
Sho Yamasaki
Researcher at Osaka University
Publications - 195
Citations - 11586
Sho Yamasaki is an academic researcher from Osaka University. The author has contributed to research in topics: C-type lectin & Immune system. The author has an hindex of 45, co-authored 156 publications receiving 9894 citations. Previous affiliations of Sho Yamasaki include Chiba University & National Institute for Medical Research.
Papers
More filters
Journal ArticleDOI
C-Type Lectin Receptor MCL Facilitates Mincle Expression and Signaling through Complex Formation
TL;DR: It is demonstrated that MCL interacts with Mincle to promote its surface expression through protein–protein interaction via its stalk region, thereby magnifying Mincle-mediated signaling.
Journal ArticleDOI
NFAM1, an immunoreceptor tyrosine-based activation motif-bearing molecule that regulates B cell development and signaling.
Makoto Ohtsuka,Hisashi Arase,Arata Takeuchi,Sho Yamasaki,Ritsuko Shiina,Tadahiro Suenaga,Daiju Sakurai,Tadashi Yokosuka,Noriko Arase,Makio Iwashima,Toshio Kitamura,Hideshige Moriya,Takashi Saito +12 more
TL;DR: The results suggest that NFAM1 modulates B cell signaling through its ITAM, which regulates B cell development.
Journal ArticleDOI
Fc receptor gamma-chain, a constitutive component of the IL-3 receptor, is required for IL-3-induced IL-4 production in basophils
Shigeaki Hida,Sho Yamasaki,Yuzuru Sakamoto,Masaya Takamoto,Kazushige Obata,Toshiyuki Takai,Hajime Karasuyama,Kazuo Sugane,Takashi Saito,Shinsuke Taki +9 more
TL;DR: It is shown that basophils lacking FcRγ developed normally and proliferated efficiently in response to interleukin 3 but were very impaired in IL-3-induced production of IL-4 and in supporting T helper type 2 differentiation.
Journal ArticleDOI
Kawasaki disease: A matter of innate immunity
Toshiro Hara,Yasutaka Nakashima,Yasunari Sakai,Hisanori Nishio,Yoshitomo Motomura,Sho Yamasaki +5 more
TL;DR: It is proposed herein that KD is an innate immune disorder resulting from the exposure of a genetically predisposed individual to microbe‐derived innate immune stimulants and that it is not a typical infectious disease.
Journal ArticleDOI
The C-type lectin receptor CLECSF8/CLEC4D is a key component of anti-mycobacterial immunity.
Gillian J. Wilson,Mohlopheni J. Marakalala,Jennifer C. Hoving,Arjan van Laarhoven,Rebecca A. Drummond,Bernhard Kerscher,Roanne Keeton,Esther van de Vosse,Tom H. M. Ottenhoff,Theo S. Plantinga,Bachti Alisjahbana,Dhirendra Govender,Gurdyal S. Besra,Mihai G. Netea,Delyth M. Reid,Janet A. Willment,Muazzam Jacobs,Sho Yamasaki,Reinout van Crevel,Gordon D. Brown,Gordon D. Brown +20 more
TL;DR: It is concluded that CLECSF8 plays a non-redundant role in anti-mycobacterial immunity in mouse and in man.