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Won Ho Kim

Researcher at Yonsei University

Publications -  748
Citations -  21405

Won Ho Kim is an academic researcher from Yonsei University. The author has contributed to research in topics: Inflammatory bowel disease & Colorectal cancer. The author has an hindex of 61, co-authored 704 publications receiving 18779 citations. Previous affiliations of Won Ho Kim include KAIST & Seoul National University.

Papers
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Journal ArticleDOI

Styrene butadiene rubber-clay nanocomposites using a latex method: morphology and mechanical properties

Won Ho Kim, +4 more
- 01 Jan 2007 - 
TL;DR: In this paper, octadecylamine modified MMT (C18-MMT) filled SBR nanocomposites were manufactured using a latex method and a compounding method.
Journal Article

Effect of ear mushroom (Auricularia) on functional constipation

Tae Il Kim, +4 more
- 01 Jul 2004 - 
TL;DR: Although the use of ear mushrooms with additives was found to be more effective on the improvement of constipation than ear mushrooms alone, it may induce abdominal discomfort or pain.
Journal ArticleDOI

Clinicopathologic Findings of Colorectal Traditional and Sessile Serrated Adenomas in Korea: A Multicenter Study

Sang Kil Lee, +23 more
- 01 Aug 2008 - 
TL;DR: Serrated polyps have emerged as important evidence supporting the serrated polyp-neoplasia pathway in colorectal carcinogenesis, an alternate to the classical adeno as mentioned in this paper.
Journal ArticleDOI

Infliximab versus Cyclosporine Treatment for Severe Corticosteroid-Refractory Ulcerative Colitis: A Korean, Retrospective, Single Center Study.

Eun Hye Kim, +6 more
- 23 Sep 2015 - 
TL;DR: Comparing the efficacy of cyclosporine and infliximab in patients with corticosteroid-refractory ulcerative colitis found inflixIMab with azathioprine may be more effective than cyclosporaine alone for preventing colectomy.
Journal ArticleDOI

The induction of STAT1 gene by activating transcription factor 3 contributes to pancreatic β-cell apoptosis and its dysfunction in streptozotocin-treated mice

Ji Yeon Kim, +7 more
- 01 Nov 2010 - 
TL;DR: It is suggested that ATF3 functions as a potent upstream regulator of STAT1 and ATF3 may play a role in STZ-induced beta-cell dysfunction by enhancing the steady state abundance ofSTAT1.