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Yuli Cai
Researcher at Wuhan University
Publications - 17
Citations - 4027
Yuli Cai is an academic researcher from Wuhan University. The author has contributed to research in topics: Fatty liver & Proinflammatory cytokine. The author has an hindex of 11, co-authored 14 publications receiving 2884 citations. Previous affiliations of Yuli Cai include Texas A&M University & Huazhong University of Science and Technology.
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Journal ArticleDOI
Association of Cardiac Injury With Mortality in Hospitalized Patients With COVID-19 in Wuhan, China.
Shaobo Shi,Mu Qin,Bo Shen,Yuli Cai,Tao Liu,Fan Yang,Wei Gong,Xu Liu,Jinjun Liang,Qinyan Zhao,He Huang,Bo Yang,Congxin Huang +12 more
TL;DR: The association between cardiac injury and mortality in patients with COVID-19 was analyzed and it was found that patients with cardiac injury had a higher proportion of multiple mottling and ground-glass opacity in radiographic findings than those without cardiac injury.
Journal ArticleDOI
Characteristics and clinical significance of myocardial injury in patients with severe coronavirus disease 2019.
Shaobo Shi,Mu Qin,Yuli Cai,Tao Liu,Bo Shen,Fan Yang,Sheng Cao,Xu Liu,Yaozu Xiang,Qinyan Zhao,He Huang,Bo Yang,Congxin Huang +12 more
TL;DR: The risk of in-hospital death among patients with severe COVID-19 can be predicted by markers of myocardial injury, and was significantly associated with senior age, inflammatory response, and cardiovascular comorbidities.
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Berberine Ameliorates Hepatic Steatosis and Suppresses Liver and Adipose Tissue Inflammation in Mice with Diet-induced Obesity.
Ting Guo,Shih Lung Woo,Xin Guo,Honggui Li,Juan Zheng,Juan Zheng,Rachel Botchlett,Mengyang Liu,Ya Pei,Hang Xu,Yuli Cai,Yuli Cai,Tianshu Zeng,Lulu Chen,Xiaodong Li,Qifu Li,Xiaoqiu Xiao,Yuqing Huo,Chaodong Wu +18 more
TL;DR: An AMP-activated protein kinase (AMPK)-independent mechanism for BBR to suppress obesity-associated inflammation and improve hepatic steatosis is revealed and actions on improving aspects of NAFLD are largely attributable to BBR suppression of inflammation, which is independent of AMPK.
Journal ArticleDOI
Disruption of adenosine 2A receptor exacerbates NAFLD through increasing inflammatory responses and SREBP1c activity
Yuli Cai,Honggui Li,Mengyang Liu,Ya Pei,Juan Zheng,Juan Zheng,Jing Zhou,Xianjun Luo,Wenya Huang,Linqiang Ma,Linqiang Ma,Qiuhua Yang,Qiuhua Yang,Shaodong Guo,Xiaoqiu Xiao,Qifu Li,Tianshu Zeng,Fanyin Meng,Heather Francis,Shannon Glaser,Lulu Chen,Yuqing Huo,Yuqing Huo,Gianfranco Alpini,Chaodong Wu +24 more
TL;DR: It is demonstrated that disruption of A2AR in both macrophage and hepatocytes accounts for increased severity of NAFLD, likely through increasing inflammation and through elevating lipogenic events due to stimulation of SREBP1c expression and transcription activity.
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Fasting blood glucose level is a predictor of mortality in patients with COVID-19 independent of diabetes history.
TL;DR: Screening FBG level is an effective method to evaluate the prognosis of patients with COVID-19 and showed FBG ≥7.0 mmol/L predicted worse outcome in hospitalized patients with co-ordinated coronavirus disease 2019 independent of diabetes history.