Zhike Lu

TL;DR: It is revealed that the inactivation of GAPDH by H2O2 induces metabolic levels of glycolysis and the PPP; the elevated levels of fructose 1,6-biphosphate (FBP) and 2-keto-3-deoxy-6-phosphogluconate (KDPG) lead to dissociation of their corresponding gly colytic repressors (GapR and HexR, respectively) from their cognate promoters,

TL;DR: In the version of this article originally published, the references were incorrectly re-ordered during production and the hyphen in “N6-methyladenosine” in the title was also superscript.

TL;DR: This corrects the article to show that the method used to derive the H2O2 “spatially aggregating force” is based on a two-step process, not a single step, like in the previous version of this paper.

TL;DR: In this paper , the authors found that neutrophil migration is necessary for antibacterial innate defense by enhancing intrinsic ability of neutrophils migration, and they found that deficiency of ALKBH5 increased mortality of mice with polymicrobial sepsis induced by cecal ligation and puncture (CLP), and Alkbh5 -deficient mice exhibited higher bacterial burden and massive proinflammatory cytokine production in the peritoneal cavity and blood because of less neutrophIL migration.

TL;DR: In the version of this Brief Communication initially published, the email address for corresponding author, Chao Xu, was incorrect and it should be listed as chaor.xu@utoronto.edu.