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Open AccessJournal ArticleDOI

A Textbook of Microbiology

R. T. Hewlett
- 01 Aug 1940 - 
- Vol. 146, Iss: 3692, pp 149-149
TLDR
This book is a text-book on general microbiology, but more than half this book is devoted to the medical aspects of the subject, little space being assigned to fermentation, soil microbiology and nitrification, micro-organisms in industry, and the like.
Abstract
SO far from being a text-book on general microbiology, as the title seems to imply, more than half this book is devoted to the medical aspects of the subject, little space being assigned to fermentation, soil microbiology and nitrification, micro-organisms in industry, and the like. A Textbook of Microbiology By Prof. Kenneth L. Burdon. Second edition of “A Textbook of Bacteriology”. Pp. xv + 638. (New York: The Macmillan Company, 1939.) 14s. net.

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Journal ArticleDOI

Molecular basis of the interaction of Salmonella with the intestinal mucosa.

TL;DR: Animal and in vitro cell culture models for the interaction of enterocolitis-causing salmonellae with the intestinal mucosa are reviewed, along with the bacterial genes that are thought to affect this interaction.
Journal ArticleDOI

The amazing potential of fungi: 50 ways we can exploit fungi industrially

Kevin D. Hyde, +69 more
- 03 Jul 2019 - 
TL;DR: This manuscript reviews fifty ways in which fungi can potentially be utilized as biotechnology and provides a flow chart that can be used to convince funding bodies of the importance of fungi for biotechnological research and as potential products.
Journal ArticleDOI

Free radicals and the etiology of colon cancer

TL;DR: Intracolonic free radical formation may explain the high incidence of cancer in the colon and rectum, compared to other regions of the GI tract, as well as the observed correlations of a higher incidence of colon cancer with red meat in the diet, which increases stool iron, and with excessive fat in the diets, which may increase the fecal content of procarcinogens and bile pigments.
Journal ArticleDOI

Oxygen radicals in ulcerative colitis.

TL;DR: This article reviews the pathophysiologic concept that superoxide and hydrogen peroxide, generated by activated leukocytes, together with low-molecular-weight chelate iron derived from fecal sources and from denatured hemoglobin, amplify the inflammatory response and subsequent mucosal damage in patients with active episodes of ulcerative colitis.