Journal ArticleDOI
Can we see the cerebellar activation effect by TMS over the back of the head
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TLDR
Information is found that antidromic corticospinal tract activation by the conditioning stimulus can produce suppression of contralateral motor cortex and several confounding factors should be excluded before concluding that the suppressive effects of ‘‘electrical cerebellar stimulation’ are derived from activation of cerebellAR structures or dis-facilitation of the dentato-thalamo-cortical pathway.About:
This article is published in Clinical Neurophysiology.The article was published on 2009-12-01. It has received 29 citations till now. The article focuses on the topics: Head (linguistics).read more
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Journal ArticleDOI
Non-invasive Cerebellar Stimulation—a Consensus Paper
Giuliana Grimaldi,Georgios P. D. Argyropoulos,A Boehringer,Pablo Celnik,Mark J. Edwards,Roberta Ferrucci,Roberta Ferrucci,Joseph M. Galea,Stefan Jun Groiss,Koichi Hiraoka,Panagiotis Kassavetis,Elise Lesage,Mario Manto,R. C. Miall,Anna Sadnicka,Yoshikazu Ugawa,Ulf Ziemann +16 more
TL;DR: There is a consensus amongst the panel of experts that both TMS and tDCS can effectively influence cerebellar functions, not only in the motor domain, with effects on visually guided tracking tasks, motor surround inhibition, motor adaptation and learning, but also for the cognitive and affective operations handled by the cerebro-cerebellar circuits.
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Human Locomotor Adaptive Learning Is Proportional to Depression of Cerebellar Excitability
TL;DR: It is demonstrated that locomotor adaptive learning in humans is proportional to cerebellar excitability depression, and this finding supports the theory that adaptive learning is mediated, at least in part, by long-term depression in Purkinje cells.
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Assessing cerebellar brain inhibition (CBI) via transcranial magnetic stimulation (TMS): A systematic review.
TL;DR: It was found that methodological design and selection of parameters in several studies may have reduced the validity of outcomes and further systematic testing of CBI protocols is warranted, both from a parameter and task‐based perspective.
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Effect of repetitive transcranial magnetic stimulation over the cerebellum on patients with ataxia after posterior circulation stroke: A pilot study.
TL;DR: It is demonstrated that 1 Hz rTMS over the cerebellum is safe, feasible and may have a beneficial effect in ataxic patients with posterior circulation stroke.
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Cerebellar Stimulation in Ataxia
TL;DR: A conditioning stimulus over the cerebellum preceding a test stimulus of the contralateral M1 enables us to study the Cerebellar regulatory functions on M1 and its usefulness as a diagnostic tool in clinical neurophysiology.
References
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Magnetic stimulation over the cerebellum in humans
TL;DR: It is concluded that magnetic stimulation over the cerebellum with a doublecone coil elicits the same suppressive effect on the motor cortex as electrical stimulation, but with less discomfort; moreover, it is believed that this effect is produced by activation of certain cerebellar structures.
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Modulation of motor cortical excitability by electrical stimulation over the cerebellum in man.
TL;DR: It is suggested that a single electrical stimulus across the base of the skull (particularly with the anode over one cerebellar hemisphere) produces a short latency (5‐6 ms) disfacilitation of the contralateral motor cortex through activation of Cerebellar structures.
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Magnetic stimulation of corticospinal pathways at the foramen magnum level in humans.
TL;DR: It is concluded that magnetic brainstem stimulation produces a single descending volley in the corticospinal tract at the foramen magnum level with less discomfort.
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Magnetic stimulation over the cerebellum in patients with ataxia
Yoshikazu Ugawa,Yasuo Terao,Ritsuko Hanajima,Katsuyuki Sakai,Toshiaki Furubayashi,Katsuyuki Machii,Ichiro Kanazawa +6 more
TL;DR: It is concluded that magnetic stimulation over the cerebellum produces the same effect as electrical stimulation even in ataxic patients, and this less painful method can be used clinically to clarify the pathomechanisms for ataxia.
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Suppression of motor cortical excitability by electrical stimulation over the cerebellum in ataxia
TL;DR: It is concluded that electrical stimulation activates cerebellar structures that suppress motor cortical excitability through a cerebellothalamocortical pathway and that the afferent systems to the cerebellum make no or little contribution to the effect.