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Evidence for the existence of monoamine neurons in the central nervous system. iv. distribution of monoamine nerve terminals in the central nervous system.

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This article is published in Acta Physiologica Scandinavica.The article was published on 1965-01-01 and is currently open access. It has received 1467 citations till now. The article focuses on the topics: Monoamine neurotransmitter & Nervous system.

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Central serotonergic projections to the nucleus tractus solitarii: Evidence from a double labeling study in the rat

TL;DR: Results provide direct evidence that brainstem serotonergic neurons contribute to the innervation of the nucleus tractus solitarii, and indicate that the nucleus raphe magnus and the nucleusRaphe dorsalis constitute two major sources of centralserotonergic projections to the nucleus tractor.
Journal Article

The coexistence of serotonin- and substance P-like immunoreactivity in the spinal cord of the rat as shown by immunofluorescent double labeling

TL;DR: The coexistence of 5-HT and SP in nerve fibers and terminals is associated with somatic and sympathetic autonomic motoneurons, and the role of these substances in somatosensation, including pain, is unclear.
Journal ArticleDOI

Cardiovascular regulation by central adrenergic mechanisms and its alteration by hypotensive drugs.

G Haeusler
- 01 Jun 1975 - 
TL;DR: Experiments following depletion of norepinephrine suggest that the central part of the baroreceptor reflex arc does not contain adrenergic neurons, but these findings are compatible with the view that some neurons within the reflex arc are supplied with α-adrenoceptors, and may represent a trigger mechanism for the initiation of hypertension.
Journal ArticleDOI

Centrally induced hypotension and bradycardia after administration of alpha-methylnoradrenaline into the area of the nucleus tractus solitarii of the rat.

TL;DR: In anaesthetized rats, bilateral injections of α‐methylnoradrenaline, noradrenalin or adrenaline into the area of the nucleus tractus solitarii of the brain stem caused dose‐dependent decreases of systemic arterial blood pressure and heart rate.
Journal ArticleDOI

Aspartic acid and glutamic acid levels in the cochlear nucleus after auditory nerve lesion

TL;DR: Ulastructural analysis of the cochlear nucleus showed that most primary auditory terminals were degenerating one day after co chlear ablation; the terminals were enlarged and the number of synaptic vesicles was reduced.
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