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Gut Microbiome and Diet

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The article was published on 2021-01-01. It has received 1 citations till now. The article focuses on the topics: Microbiome & Biology.

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Additional file 1: of An expansion of rare lineage intestinal microbes characterizes rheumatoid arthritis

TL;DR: In this article, the 16S ribosomal DNA of rheumatoid arthritis patients, first-degree relatives, and random healthy non-RA controls were sequenced to define a microbial and metabolite profile that could predict disease status.
References
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Journal ArticleDOI

Dietary Modulation of the Human Colonic Microbiota: Introducing the Concept of Prebiotics

TL;DR: By combining the rationale of pro- and prebiotics, the concept of synbiotics is proposed to characterize some colonic foods with interesting nutritional properties that make these compounds candidates for classification as health-enhancing functional food ingredients.
Journal ArticleDOI

Diet rapidly and reproducibly alters the human gut microbiome

TL;DR: Increases in the abundance and activity of Bilophila wadsworthia on the animal-based diet support a link between dietary fat, bile acids and the outgrowth of microorganisms capable of triggering inflammatory bowel disease.
Journal ArticleDOI

Impact of diet in shaping gut microbiota revealed by a comparative study in children from Europe and rural Africa

TL;DR: In this paper, the authors compared the fecal microbiota of European children (EU) and that of children from a rural African village of Burkina Faso (BF), where the diet, high in fiber content, is similar to that of early human settlements at the time of the birth of agriculture.
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Changes in gut microbiota control metabolic endotoxemia-induced inflammation in high-fat diet-induced obesity and diabetes in mice

TL;DR: It is found that changes of gut microbiota induced by an antibiotic treatment reduced metabolic endotoxemia and the cecal content of LPS in both high-fat–fed and ob/ob mice, demonstrating that changes in gut microbiota controls metabolic endotoxinemia, inflammation, and associated disorders by a mechanism that could increase intestinal permeability.