Low-energy impact of human cartilage: predictors for microcracking the network of collagen
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TLDR
The minimum mechanical impact to cause microstructural damage in the network of collagen (microcracking) within human cartilage and hypothesized that energies below 0.1 J or 1 mJ/mm3 would suffice, found that impact energy/energy dissipation density and nominal stress/stress rate were significant predictors of microcracking.About:
This article is published in Osteoarthritis and Cartilage.The article was published on 2017-04-01 and is currently open access. It has received 22 citations till now.read more
Citations
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Compositional and Metabolic Changes in Damaged Cartilage are Peak-Stress, Stress-Rate, and Loading-Duration Dependent
TL;DR: The results showed that damage to cartilage required repeated impacts with a peak stress of at least 2.5 MPa and a stress rate of at at least 30 MPa/sec for 2 minutes or longer, suggesting that impact damage is cumulative and stress-rate dependent.
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A review of impact resistant biological and bioinspired materials and structures
TL;DR: In this paper, a review of impact resistant biological systems with a focus on their recurrent structural design elements, material properties, and energy absorbing mechanisms is presented, and the impact resistant structures at the micro- and meso-scales are classified into layered, gradient, tubular, sandwich and sutured.
Journal ArticleDOI
Visceral pain from colon and rectum: the mechanotransduction and biomechanics.
Bin Feng,Tiantian Guo +1 more
TL;DR: This review calls for focused attention on targeting colorectal mechanotransduction as a new strategy for managing visceral pain, which can also have an added benefit of limited CNS side effects, because mechanotranduction arises from peripheral organs.
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Comparison between in vitro and in vivo cartilage overloading studies based on a systematic literature review
TL;DR: The rationale of the current review was to identify consistencies and inconsistencies between in vitro and in vivo studies on mechanically‐induced structural damage in articular cartilage, such that variables worth interesting to further explore using either one of these approaches can be identified.
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Cartilage-on-cartilage cyclic loading induces mechanical and structural damage.
TL;DR: It is demonstrated that with increased number of cycles, cartilage undergoes both tissue softening and structural damage, suggesting that cyclic loading can cause in vivo damage.
References
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Interspecies comparisons of in situ intrinsic mechanical properties of distal femoral cartilage
Kyriacos A. Athanasiou,Melvin P. Rosenwasser,Joseph A. Buckwalter,Theodore I. Malinin,Van C. Mow +4 more
TL;DR: The results lead to the conclusion that patellar groove cartilage can undergo greater and faster compression under high compressive loads and can more rapidly compress to create a congruent patellofemoral joint articulation.
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Biophysical chemistry of cartilaginous tissues with special reference to solute and fluid transport.
Journal ArticleDOI
Survival of articular cartilage after controlled impact
TL;DR: The data suggest that impact loads sufficient to fracture a femoral shaft of an automobile occupant are nearly sufficient to cause chondrocyte death and fissuring in the articular cartilage of either the knee or the hip if the load-bearing areas measure less than 500 square millimeters.
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The extent of matrix damage and chondrocyte death in mechanically traumatized articular cartilage explants depends on rate of loading
TL;DR: It is indicated that the rate of loading can significantly affect the degree of matrix damage, the distribution of dead cells, and the amount of cell death in unconfined compression experiments on explants of articular cartilage.
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