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Deepali Gupta

Researcher at University of Texas Southwestern Medical Center

Publications -  52
Citations -  1064

Deepali Gupta is an academic researcher from University of Texas Southwestern Medical Center. The author has contributed to research in topics: Elevated plus maze & Ghrelin. The author has an hindex of 17, co-authored 47 publications receiving 827 citations. Previous affiliations of Deepali Gupta include Jaypee Institute of Information Technology & Post Graduate Institute of Medical Education and Research.

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The energetic brain – A review from students to students

Melina Paula Bordone, +38 more
TL;DR: This review illustrates that the vertebrate brain has a high need for energy because of the high number of neurons and the need to maintain a delicate interplay between energy metabolism, neurotransmission, and plasticity.
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Antidepressant effects of insulin in streptozotocin induced diabetic mice: Modulation of brain serotonin system.

TL;DR: The data indicate that insulin exhibits antidepressant effects in depression associated with STZ induced diabetes in mice through the elevation of the brain serotonin levels.
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Fine needle aspiration cytology in the diagnosis of tuberculous mastitis.

TL;DR: The diagnosis of granulomatous mastitis must be made with caution, even in the absence of AFB, only after a sufficient trial of antituberculosis treatment has been given and the patient fails to respond should an alternative diagnosis be suggested.
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Diabetes-associated depression: The serotonergic system as a novel multifunctional target

TL;DR: The main purpose of this review was to address the sophisticated mechanisms of this comorbidity with a view of developing potential novel targets with higher efficacy and specificity.
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Effect of a novel 5-HT3 receptor antagonist 4i, in corticosterone-induced depression-like behavior and oxidative stress in mice

TL;DR: The study suggests that 5HT3 antagonist; 4i may be implicated as pharmacological intervention targeting depressive-like anomaly associated with HPA-axis dysregulation, which may plausibly demonstrate one of the key mechanisms for antidepressant-like effects of the compounds.